Alex Tarnava

It regulates our glucose homeostasis and our insulin response.

And actually what you see in healthy people, when you're hungry, you have quite high ghrelin.

That's the hunger cycle.

And then when you eat, it drops to nothing.

But in chronically obese people, it's just kind of flatlined in between.

That's right.

Right.

And something that similar happens with GLP-1 too.

Actually, it's similar to actually how insulin works.

So hydrogen has shown to restore those peaks and valleys of grout.

Now with GLP-1, we showed in a rodent research where we induced obesity through a high fat diet.

Yeah.

That what we know with GLP-1 in early phases of obesity is it goes into hyperdrive, trying to overcorrect, but then the feedback mechanism breaks and then we don't get any GLP-1 secretion.

It's just always chronically low.

Yeah.

Same thing with like insulin response going into diabetes.

So the hydrogen is

Actually, and this is just in rats, right?

So it's just in rodents, so not humans, but we actually attenuated the false rise in GLP-1 in inducing obesity, but yet

the hydrogen group was the only one that resisted weight gain.