Christian Drapeau

If we look at the development of it, I think the mechanism of action is that atherosclerosis starts with a microlesion in your arteries. If it does not repair, then it gets infiltrated by immune cells, who then become foam cells or like dysregulated cells because of certain fat ingestion.

If we look at the development of it, I think the mechanism of action is that atherosclerosis starts with a microlesion in your arteries. If it does not repair, then it gets infiltrated by immune cells, who then become foam cells or like dysregulated cells because of certain fat ingestion.

then they start, in this dysregulation, they start to trigger inflammation, calcification, and then the whole process builds up into atherosclerosis. But if you have enough stem cells to repair the microlesions at day one, then none of that process follows. So I think that the link here, and there is, like you take atherosclerosis, you take, for example...

then they start, in this dysregulation, they start to trigger inflammation, calcification, and then the whole process builds up into atherosclerosis. But if you have enough stem cells to repair the microlesions at day one, then none of that process follows. So I think that the link here, and there is, like you take atherosclerosis, you take, for example...

One sign, one tool to do, what is the word here? Like, not a survey, but like a precursor sign of the development of atherosclerosis is a thickening of the basal membrane, if you want, of your arteries. the intima media layer. A precursor sign to atherosclerosis is the thickness of the wall of the arteries.

One sign, one tool to do, what is the word here? Like, not a survey, but like a precursor sign of the development of atherosclerosis is a thickening of the basal membrane, if you want, of your arteries. the intima media layer. A precursor sign to atherosclerosis is the thickness of the wall of the arteries.

So you can look right now that if the difference between somebody that is below the thickness, like which is healthy,

So you can look right now that if the difference between somebody that is below the thickness, like which is healthy,

or above the thickness that is basically telling you you are now developing atherosclerosis, and you can see that there's a difference in the number of stem cells in these individuals, meaning that you having low level of stem cells is already a sign that you're in line to develop atherosclerosis.

or above the thickness that is basically telling you you are now developing atherosclerosis, and you can see that there's a difference in the number of stem cells in these individuals, meaning that you having low level of stem cells is already a sign that you're in line to develop atherosclerosis.

And if you look at all the associated problems associated to atherosclerosis, like there's a much higher incidence of erectile dysfunction, for example, with people with atherosclerosis. So same thing. People with erectile dysfunction have fewer stem cells in circulation. So there's a link here. Well documented.

And if you look at all the associated problems associated to atherosclerosis, like there's a much higher incidence of erectile dysfunction, for example, with people with atherosclerosis. So same thing. People with erectile dysfunction have fewer stem cells in circulation. So there's a link here. Well documented.

If you are a poor mobilizer with fewer stem cells in circulation, you are likely to develop atherosclerosis.

If you are a poor mobilizer with fewer stem cells in circulation, you are likely to develop atherosclerosis.

Well, the only thing I can say is that the scientific literature is clear on one thing. And it's the fact that while you may have high level of lipoprotein A, That itself is not the issue. The issue is that it compounds with the microlesions, you know, what I talked about before, that then makes the problem much more risky for you, risky to develop.

Well, the only thing I can say is that the scientific literature is clear on one thing. And it's the fact that while you may have high level of lipoprotein A, That itself is not the issue. The issue is that it compounds with the microlesions, you know, what I talked about before, that then makes the problem much more risky for you, risky to develop.

But if you boost the repair, then the lipoprotein A is no longer an issue in the same way. So I would say your score in terms of the health of your cardiovascular system will definitely improve. And that's tied to the stem cells. So when I get these tests, I got these tests done maybe like three years ago. And I'm 60 in a few weeks. And I have the cardiovascular system of like a 30-year-old.

But if you boost the repair, then the lipoprotein A is no longer an issue in the same way. So I would say your score in terms of the health of your cardiovascular system will definitely improve. And that's tied to the stem cells. So when I get these tests, I got these tests done maybe like three years ago. And I'm 60 in a few weeks. And I have the cardiovascular system of like a 30-year-old.

And I've been taking these products for a long time. And I attribute it to the fact that I have more stem cells to just repair. I'm not aware of an effect of the stem cells directly on lipoprotein A. I did some research and I could not find anything. However, what I can say is that I chose these ingredients and I put them together for their effect on stem cells.

And I've been taking these products for a long time. And I attribute it to the fact that I have more stem cells to just repair. I'm not aware of an effect of the stem cells directly on lipoprotein A. I did some research and I could not find anything. However, what I can say is that I chose these ingredients and I put them together for their effect on stem cells.