Dr. Adeel Khan

Where does that foreign DNA come from? It comes from E. coli. So we're not injecting bacteria into your body, but we're injecting something called plasmid. The plasmid is kind of think of it like exchanging cards with people. It's how bacteria exchange information. So we're isolating that plasmid from the E. coli, and then we're using that as a vector.

Where does that foreign DNA come from? It comes from E. coli. So we're not injecting bacteria into your body, but we're injecting something called plasmid. The plasmid is kind of think of it like exchanging cards with people. It's how bacteria exchange information. So we're isolating that plasmid from the E. coli, and then we're using that as a vector.

So plasmids have been around in microbiology for two, three decades. They're not new, but the new breakthrough was just getting the plasmid to not shut off. Because normally when you put a plasmid in the human body, your cell does something called transient silencing, which just says, hey, this doesn't belong here. I'm going to shut you off. And then it just shuts it off.

So plasmids have been around in microbiology for two, three decades. They're not new, but the new breakthrough was just getting the plasmid to not shut off. Because normally when you put a plasmid in the human body, your cell does something called transient silencing, which just says, hey, this doesn't belong here. I'm going to shut you off. And then it just shuts it off.

And then but we figured out a way for it to keep remain on specifically to express whatever gene of interest we want, which obviously are longevity genes. We're not going to put something in your body that's not useful. We're going to put something in your body that's going to have some real longevity benefits. And polystatin has been studied again for two decades.

And then but we figured out a way for it to keep remain on specifically to express whatever gene of interest we want, which obviously are longevity genes. We're not going to put something in your body that's not useful. We're going to put something in your body that's going to have some real longevity benefits. And polystatin has been studied again for two decades.

Polystatin is something your body makes. It's basically a way for your body to put on more muscle because the higher your polystatin, the lower your myostatin. which is basically imagine the brakes on your muscle. And so if you take the brakes off a little bit, it's not like you're going to get jacked like the anabolic steroids, but it will make it a little bit easier to put on muscle.

Polystatin is something your body makes. It's basically a way for your body to put on more muscle because the higher your polystatin, the lower your myostatin. which is basically imagine the brakes on your muscle. And so if you take the brakes off a little bit, it's not like you're going to get jacked like the anabolic steroids, but it will make it a little bit easier to put on muscle.

But much more importantly, and this is why I like polystatin, it's very anti-catabolic. Because as you get older, especially after age 60, your catabolism increases exponentially.

But much more importantly, and this is why I like polystatin, it's very anti-catabolic. Because as you get older, especially after age 60, your catabolism increases exponentially.

There's anabolism and catabolism. Anabolism is building more tissue. Catabolism is what happens with aging, which is loss of tissue. We actually lose, I forgot the exact percentage, but we lose some like, you know, very large percentage of our total tissue mass as we age. And that's loss of mitochondria, that's loss of muscle, that's loss of bone density. So we lose a lot of tissue as we age.

There's anabolism and catabolism. Anabolism is building more tissue. Catabolism is what happens with aging, which is loss of tissue. We actually lose, I forgot the exact percentage, but we lose some like, you know, very large percentage of our total tissue mass as we age. And that's loss of mitochondria, that's loss of muscle, that's loss of bone density. So we lose a lot of tissue as we age.

And so if you can maintain as much tissue as possible, that's going to be a a net positive thing, and especially muscle tissue, because muscle tissue is the most metabolically active. It has all these protective cytokines that are released called myokines that turn off tumor suppressors that help prevent cancer, help with diabetes. I'm sure most people know about the benefits of muscle.

And so if you can maintain as much tissue as possible, that's going to be a a net positive thing, and especially muscle tissue, because muscle tissue is the most metabolically active. It has all these protective cytokines that are released called myokines that turn off tumor suppressors that help prevent cancer, help with diabetes. I'm sure most people know about the benefits of muscle.

and anything we can do to help preserve that, I think it's going to have a net positive effect on your health. There's obviously a lot of people get concerned. They're like, how do you know this is a hundred percent safe? Because it's new. It is new, obviously. And I'm not saying there's not enough long-term data to know, is it going to do something to you 20 years from now?

and anything we can do to help preserve that, I think it's going to have a net positive effect on your health. There's obviously a lot of people get concerned. They're like, how do you know this is a hundred percent safe? Because it's new. It is new, obviously. And I'm not saying there's not enough long-term data to know, is it going to do something to you 20 years from now?

We can't say that for sure, but what I can say, and the reason I'm a believer in it and the reason I've done it for myself and obviously patients and Many, I've injected hundreds of people with it.

We can't say that for sure, but what I can say, and the reason I'm a believer in it and the reason I've done it for myself and obviously patients and Many, I've injected hundreds of people with it.

And the reason is because, and the reason patients like it too, is because the net positive of having more muscle and having reduced systemic inflammation, which is what false statin does, to me is going to outweigh any theoretical risk that maybe we don't know about. But I think it's unlikely anyway, because we understand how false statin works.

And the reason is because, and the reason patients like it too, is because the net positive of having more muscle and having reduced systemic inflammation, which is what false statin does, to me is going to outweigh any theoretical risk that maybe we don't know about. But I think it's unlikely anyway, because we understand how false statin works.