Dr. Jordan Vaughn

Yeah, and not just that. The unique thing, and that's what Richard Petorius' work out of Stellenbosch in South Africa proved, was that the S1 subunit, the same one that we can talk about in this paper related to Parkinson's, actually is able to elicit fibrinogen to turn into fibrin without thrombin. And I know that sounds like a fancy thing, but basically itself can actually make a clot form.

Yeah, and not just that. The unique thing, and that's what Richard Petorius' work out of Stellenbosch in South Africa proved, was that the S1 subunit, the same one that we can talk about in this paper related to Parkinson's, actually is able to elicit fibrinogen to turn into fibrin without thrombin. And I know that sounds like a fancy thing, but basically itself can actually make a clot form.

And we had really never seen anything quite like that before. And that's another reason why we see a lot of young people with strokes in areas of the brain that aren't typical. And one of the reasons is because the concept of what we call microthrombosis in situ. And that's a fancy word for saying the clot didn't come from somewhere. It formed right there in the small vessel.

And we had really never seen anything quite like that before. And that's another reason why we see a lot of young people with strokes in areas of the brain that aren't typical. And one of the reasons is because the concept of what we call microthrombosis in situ. And that's a fancy word for saying the clot didn't come from somewhere. It formed right there in the small vessel.

And that's why you're having the issue because you just got a vaccine or you just had COVID.

And that's why you're having the issue because you just got a vaccine or you just had COVID.

I will say in my subset, again, I've seen about 4,000 plus people. And, you know, probably 80% of them are vaccinated and COVID, obviously. I have about 20%. Again, I'm in Alabama. So 20% in Alabama is, you know, there are people that have never had the vaccine that have long COVID because, again, the spike protein is a pathogen.

I will say in my subset, again, I've seen about 4,000 plus people. And, you know, probably 80% of them are vaccinated and COVID, obviously. I have about 20%. Again, I'm in Alabama. So 20% in Alabama is, you know, there are people that have never had the vaccine that have long COVID because, again, the spike protein is a pathogen.

But in my case, there are also plenty of people that I have, probably maybe a couple hundred, that still don't have nucleocapsid. and they have injury. Does that make sense? So it's truly a nucleocapsid antibody. So basically, the vaccine is their injury.

But in my case, there are also plenty of people that I have, probably maybe a couple hundred, that still don't have nucleocapsid. and they have injury. Does that make sense? So it's truly a nucleocapsid antibody. So basically, the vaccine is their injury.

Interesting. Yeah, the interesting thing that Jacques Lobsher saw, he's a cardiac intensivist in South Africa, during the acute disease was that this fibrin was resistant to being broken down. So resistant to fibrinolysis. So not only does the spike protein make fibrin, it makes it in a way that is abnormal. And so it's amyloid. That's why I'm able to see it under the microscope when I stain it.

Interesting. Yeah, the interesting thing that Jacques Lobsher saw, he's a cardiac intensivist in South Africa, during the acute disease was that this fibrin was resistant to being broken down. So resistant to fibrinolysis. So not only does the spike protein make fibrin, it makes it in a way that is abnormal. And so it's amyloid. That's why I'm able to see it under the microscope when I stain it.

for diaflavin t and you know i kind of look at it from a simple perspective you know normal fibrin we want the body to make it we need to clot when we have kind of an area that needs to be uh stopped from bleeding but that fibrin kind of forms like spaghetti that just came out of the colander that you can pull apart but instead the fibrin that forms in reaction to the s1 subunit of the spike protein is kind of like burnt spaghetti casserole that you have to get a brillo pad to get off the casserole dish and a lot of these people

for diaflavin t and you know i kind of look at it from a simple perspective you know normal fibrin we want the body to make it we need to clot when we have kind of an area that needs to be uh stopped from bleeding but that fibrin kind of forms like spaghetti that just came out of the colander that you can pull apart but instead the fibrin that forms in reaction to the s1 subunit of the spike protein is kind of like burnt spaghetti casserole that you have to get a brillo pad to get off the casserole dish and a lot of these people

the issues are related to their ability to fibrinolyze or their ability to break it down. So some of these people don't even have, a lot of my patients do have underlying thrombophilic or clotting disorders that they never knew about and the vaccine or COVID elicited it. But some of them don't have that. Instead, they have fibrinolytic disorders. Their ability to break down fibrin is compromised.

the issues are related to their ability to fibrinolyze or their ability to break it down. So some of these people don't even have, a lot of my patients do have underlying thrombophilic or clotting disorders that they never knew about and the vaccine or COVID elicited it. But some of them don't have that. Instead, they have fibrinolytic disorders. Their ability to break down fibrin is compromised.

And that is a kind of a, it's actually fairly common in Anglo-European patients One of the things that we look for is called plasminogen activator inhibitor, which is basically something that tells the body, hey, stop breaking this down. And COVID causes you to overexpress it. The spike protein does too. And that makes your body, hey, you've got this clot here. Your body should break it down.

And that is a kind of a, it's actually fairly common in Anglo-European patients One of the things that we look for is called plasminogen activator inhibitor, which is basically something that tells the body, hey, stop breaking this down. And COVID causes you to overexpress it. The spike protein does too. And that makes your body, hey, you've got this clot here. Your body should break it down.

It's already harder for your body to break it down. And we're going to give it a signal that says don't do it.

It's already harder for your body to break it down. And we're going to give it a signal that says don't do it.