Dr. Jordan Vaughn
speaker
82 appearances
1 recordings
1 series
first heard Jun 2025
last heard Jun 2025
Dr. Jordan Vaughn’s voice in public audio — every appearance, attributed to the second.
Trend
recordings per month · last 12 monthsNo recordings in the last 12 months.Older appearances are listed below; set an alert to hear about the next one.
Appearances
Yeah, and not just that. The unique thing, and that's what Richard Petorius' work out of Stellenbosch in South Africa proved, was that the S1 subunit, the same one that we can talk about in this paper related to Parkinson's, actually is able to elicit fibrinogen to turn into fibrin without thrombin. And I know that sounds like a fancy thing, but basically itself can actually make a clot form.
And we had really never seen anything quite like that before. And that's another reason why we see a lot of young people with strokes in areas of the brain that aren't typical. And one of the reasons is because the concept of what we call microthrombosis in situ. And that's a fancy word for saying the clot didn't come from somewhere. It formed right there in the small vessel.
And that's why you're having the issue because you just got a vaccine or you just had COVID.
I will say in my subset, again, I've seen about 4,000 plus people. And, you know, probably 80% of them are vaccinated and COVID, obviously. I have about 20%. Again, I'm in Alabama. So 20% in Alabama is, you know, there are people that have never had the vaccine that have long COVID because, again, the spike protein is a pathogen.
But in my case, there are also plenty of people that I have, probably maybe a couple hundred, that still don't have nucleocapsid. and they have injury. Does that make sense? So it's truly a nucleocapsid antibody. So basically, the vaccine is their injury.
Interesting. Yeah, the interesting thing that Jacques Lobsher saw, he's a cardiac intensivist in South Africa, during the acute disease was that this fibrin was resistant to being broken down. So resistant to fibrinolysis. So not only does the spike protein make fibrin, it makes it in a way that is abnormal. And so it's amyloid. That's why I'm able to see it under the microscope when I stain it.
for diaflavin t and you know i kind of look at it from a simple perspective you know normal fibrin we want the body to make it we need to clot when we have kind of an area that needs to be uh stopped from bleeding but that fibrin kind of forms like spaghetti that just came out of the colander that you can pull apart but instead the fibrin that forms in reaction to the s1 subunit of the spike protein is kind of like burnt spaghetti casserole that you have to get a brillo pad to get off the casserole dish and a lot of these people
the issues are related to their ability to fibrinolyze or their ability to break it down. So some of these people don't even have, a lot of my patients do have underlying thrombophilic or clotting disorders that they never knew about and the vaccine or COVID elicited it. But some of them don't have that. Instead, they have fibrinolytic disorders. Their ability to break down fibrin is compromised.
And that is a kind of a, it's actually fairly common in Anglo-European patients One of the things that we look for is called plasminogen activator inhibitor, which is basically something that tells the body, hey, stop breaking this down. And COVID causes you to overexpress it. The spike protein does too. And that makes your body, hey, you've got this clot here. Your body should break it down.
It's already harder for your body to break it down. And we're going to give it a signal that says don't do it.
So a lot of what we use are things that actually kind of tilt the balance toward fibrinolysis. So we inhibit the production or repair of fibrin. So basically dual anti-platelets are a lot of times what we use. So Jocko's original kind of theory was triple anticoagulant, basically triple therapy is what we would have called it.
But it's basically attacking not only the platelets, but also inhibiting the fibrin production, then allowing the body- Wait, wait, why would the platelet adhesion affect fibrin activation? Sure.
the spike protein also irreversibly activates platelets okay so you've got a nasty spaghetti soup uh that uh not only the building blocks of the clot like the platelets but also the mortar is sticky as heck yeah and and i i i know dr mccullough is very hot on using natto kinase and i think he thinks he's going at a similar mechanism do you feel out where that works
Yeah, so natokinase is a natural fibrinolytic. So it's basically kind of speeding up the Pac-Man's to break it down. The only downside to that, and that's what we have found in looking at like the proteomics of the fibrin, is that it is full of nasty crap. And so sometimes when you take that, you're going to actually elicit an inflammatory response.
So a lot of what we do is put people on things that kind of naturally help you fibrinolyze and then actually calm down the reaction with things like antihistamines and other mast cell stabilizing agents and then kind of pursue fibrinolytics. Does that make sense?
Yeah, aspirin, Eliquis, those kind of things. I also use a lot of what we call pentoxyfilene, which is a rheological agent that helps red blood cells change. It's an old one. A lot of the stuff we're using, that's the beauty of actually using your brain.
Yeah, to me, there's no question that the vaccine has no use in today's world. Even then, as they update it for the fall, first of all, even if it didn't have negative and deadly consequences, it's not going to be effective by the time they roll it out anyway, just because of the mutation of the virus. So it's literally the craziest thing I've ever seen.
It's like literally playing whack-a-mole and you're always going to be behind. And so there is no benefit from that standpoint. The other thing that I do is I really try to improve the immune system because Because one of the things we found is the ability to actually fight and downregulate the abnormal immune reaction is going to be based on your overall health.
And so a lot of things like vitamin D, you know, zinc, all these things that are probably deficient in our diets anyway. Plus, we sit inside and don't get enough sun. I mean, our bodies are kind of ready for an attack. And so a lot of that is just basically good health. And then the last thing is I do use a lot of natokinase. I use like things like pycnogenol, which is French maritime tree bark.
I use things like diosmin, which is a flavonoid, quercetin. There's a lot of things that are actually very good for the vessels. Good anyway. I mean, I take them every day just as a person. The more I read about this stuff, the bigger my supplement pile actually gets when I take in the morning.
Showing 21–40 of 82 · page 2 of 5
← Previous
Next →