Dr. Spencer Nadolsky

And I was like, yeah, that's pretty much it. And I don't even blame... I was just on another podcast. I don't even get mad at these people because I had it myself. I was a young wrestler before. pretty jacked dude and been like, why, why aren't these people just like eating better and exercising? Why? It seems like it's just a, they're lazy and gluttonous or something like that.

It wasn't until, you know, going through med school and then going to conferences, talking to very smart neurobiology experts and researchers and other obesity doctors, and then talking to patients and really seeing the pattern where I started to understand it. And actually it was when I did a bodybuilding competition and got a that brought my calories just to 20.

It wasn't until, you know, going through med school and then going to conferences, talking to very smart neurobiology experts and researchers and other obesity doctors, and then talking to patients and really seeing the pattern where I started to understand it. And actually it was when I did a bodybuilding competition and got a that brought my calories just to 20.

It wasn't until, you know, going through med school and then going to conferences, talking to very smart neurobiology experts and researchers and other obesity doctors, and then talking to patients and really seeing the pattern where I started to understand it. And actually it was when I did a bodybuilding competition and got a that brought my calories just to 20.

I'm trying to eat 2,200 calories, which is probably what most people would be like, that seems like a lot. I was starving on 2,200 calories. And I was, I, I couldn't stop myself from eating Pringles. And like, I like started kind of having disordered eating. Like I couldn't, I couldn't stop. I was like, I was so ravenously hungry and craving. And I, and what my patients described was kind of that.

I'm trying to eat 2,200 calories, which is probably what most people would be like, that seems like a lot. I was starving on 2,200 calories. And I was, I, I couldn't stop myself from eating Pringles. And like, I like started kind of having disordered eating. Like I couldn't, I couldn't stop. I was like, I was so ravenously hungry and craving. And I, and what my patients described was kind of that.

I'm trying to eat 2,200 calories, which is probably what most people would be like, that seems like a lot. I was starving on 2,200 calories. And I was, I, I couldn't stop myself from eating Pringles. And like, I like started kind of having disordered eating. Like I couldn't, I couldn't stop. I was like, I was so ravenously hungry and craving. And I, and what my patients described was kind of that.

So I could imagine them being you know, in a 300 pound or 250 pound body with a lot of excess adipose tissue, trying to lose weight and feeling that way, just that, you know, I was already really lean, but like they're feeling that way at a higher body fat. That's what made me go like, oh man, I think I missed the mark.

So I could imagine them being you know, in a 300 pound or 250 pound body with a lot of excess adipose tissue, trying to lose weight and feeling that way, just that, you know, I was already really lean, but like they're feeling that way at a higher body fat. That's what made me go like, oh man, I think I missed the mark.

So I could imagine them being you know, in a 300 pound or 250 pound body with a lot of excess adipose tissue, trying to lose weight and feeling that way, just that, you know, I was already really lean, but like they're feeling that way at a higher body fat. That's what made me go like, oh man, I think I missed the mark.

And so there's two different things because people think of the disease in two different ways. The disease, the way the AMA kind of described it back in 2013, the criteria of like certain signs and symptoms, certain associated with morbidity, mortality, those types of things, like definition of disease. a disease versus the pathophysiology of what drives it.

And so there's two different things because people think of the disease in two different ways. The disease, the way the AMA kind of described it back in 2013, the criteria of like certain signs and symptoms, certain associated with morbidity, mortality, those types of things, like definition of disease. a disease versus the pathophysiology of what drives it.

And so there's two different things because people think of the disease in two different ways. The disease, the way the AMA kind of described it back in 2013, the criteria of like certain signs and symptoms, certain associated with morbidity, mortality, those types of things, like definition of disease. a disease versus the pathophysiology of what drives it.

So right now, there's the Lancet Commission just came out with their whole definition of obesity, and they tried to describe basically preclinical obesity. So basically people that don't have those sequelae or issues from their excess adipose tissue, they're healthy, basically, other than just a lot of adipose tissue.

So right now, there's the Lancet Commission just came out with their whole definition of obesity, and they tried to describe basically preclinical obesity. So basically people that don't have those sequelae or issues from their excess adipose tissue, they're healthy, basically, other than just a lot of adipose tissue.

So right now, there's the Lancet Commission just came out with their whole definition of obesity, and they tried to describe basically preclinical obesity. So basically people that don't have those sequelae or issues from their excess adipose tissue, they're healthy, basically, other than just a lot of adipose tissue.

Then they talk about clinical obesity and that those are people with, say, like type 2 diabetes or prediabetes, hypertension, some issues arising from the excess adipose tissue. So that's how some people talk about the disease of obesity. But what I think you're getting at and what I like to talk about is that the pathophysiology of what drives that adipose gain. Why is it that some people

Then they talk about clinical obesity and that those are people with, say, like type 2 diabetes or prediabetes, hypertension, some issues arising from the excess adipose tissue. So that's how some people talk about the disease of obesity. But what I think you're getting at and what I like to talk about is that the pathophysiology of what drives that adipose gain. Why is it that some people

Then they talk about clinical obesity and that those are people with, say, like type 2 diabetes or prediabetes, hypertension, some issues arising from the excess adipose tissue. So that's how some people talk about the disease of obesity. But what I think you're getting at and what I like to talk about is that the pathophysiology of what drives that adipose gain. Why is it that some people

you know, they just change their habits a little bit and lose the weight and never gain it back versus other people. They try to lose the weight and their body fights them tooth and nail and they essentially just cannot lose the weight or keep it off without some extra tool despite their best efforts and all the the amount of effort that they put into it.