Dr. Teo Soleymani

So why I bring this up and why we talk about sunscreens and barriers to blockade first, topical sunscreens are not the only form. In fact, clothing and shields, shade and hat are, in my opinion, better. And a lot of studies suggest that they're better. There are oral supplements that can also protect your skin from burning and reduce your skin cancer risk.

And number three, not everything is sun-driven. We do know that diligent sunscreen use decreases the incidence or the amount of melanoma formation and squamous cell formation, but we still don't know whether that reduction has changed the number of patients dying from that. We also know that it has no bearing in the development of the most common skin cancer, which is basal cells.

And number three, not everything is sun-driven. We do know that diligent sunscreen use decreases the incidence or the amount of melanoma formation and squamous cell formation, but we still don't know whether that reduction has changed the number of patients dying from that. We also know that it has no bearing in the development of the most common skin cancer, which is basal cells.

And number three, not everything is sun-driven. We do know that diligent sunscreen use decreases the incidence or the amount of melanoma formation and squamous cell formation, but we still don't know whether that reduction has changed the number of patients dying from that. We also know that it has no bearing in the development of the most common skin cancer, which is basal cells.

So we go back to the drawing board and scratch our heads saying, hey, what is the trigger? You know, the genetics loads the gun, for example, and maybe even pulls the hammer. But what in the environment is the trigger? Is it strictly UV? Is it some other culprit? So sunscreens are a really hotly debated topic. So many things to talk about in terms of sunscreen.

So we go back to the drawing board and scratch our heads saying, hey, what is the trigger? You know, the genetics loads the gun, for example, and maybe even pulls the hammer. But what in the environment is the trigger? Is it strictly UV? Is it some other culprit? So sunscreens are a really hotly debated topic. So many things to talk about in terms of sunscreen.

So we go back to the drawing board and scratch our heads saying, hey, what is the trigger? You know, the genetics loads the gun, for example, and maybe even pulls the hammer. But what in the environment is the trigger? Is it strictly UV? Is it some other culprit? So sunscreens are a really hotly debated topic. So many things to talk about in terms of sunscreen.

Great question. And the more we dive into this and the more experts we look at and the more data we collect, the more we're scratching our head as to we don't know and why this occurs. Now, I'm not saying don't use sunscreen. Let's just let that be known. I think sunscreens are excellent forms to protect against premature photoaging and signs of sun damage.

Great question. And the more we dive into this and the more experts we look at and the more data we collect, the more we're scratching our head as to we don't know and why this occurs. Now, I'm not saying don't use sunscreen. Let's just let that be known. I think sunscreens are excellent forms to protect against premature photoaging and signs of sun damage.

Great question. And the more we dive into this and the more experts we look at and the more data we collect, the more we're scratching our head as to we don't know and why this occurs. Now, I'm not saying don't use sunscreen. Let's just let that be known. I think sunscreens are excellent forms to protect against premature photoaging and signs of sun damage.

I think sunscreens help reduce the incidence of common garden variety skin cancers, but they are not the only form of protection, and they seem to not be as important or have as much effect in reducing the incidence of our most common skin cancer, which is basal cell carcinoma. Moreover, I would say, and the data unfortunately shows,

I think sunscreens help reduce the incidence of common garden variety skin cancers, but they are not the only form of protection, and they seem to not be as important or have as much effect in reducing the incidence of our most common skin cancer, which is basal cell carcinoma. Moreover, I would say, and the data unfortunately shows,

I think sunscreens help reduce the incidence of common garden variety skin cancers, but they are not the only form of protection, and they seem to not be as important or have as much effect in reducing the incidence of our most common skin cancer, which is basal cell carcinoma. Moreover, I would say, and the data unfortunately shows,

that the majority of skin cancers that we see that end up hurting people or killing patients don't arise in chronic sun-exposed areas to begin with. I have a 27-year-old patient right now dying of a metastatic melanoma that arose in completely sun protected skin. I have a 56-year-old mechanic right now who's dying of a squamous cell carcinoma that arose from the back of his ear.

that the majority of skin cancers that we see that end up hurting people or killing patients don't arise in chronic sun-exposed areas to begin with. I have a 27-year-old patient right now dying of a metastatic melanoma that arose in completely sun protected skin. I have a 56-year-old mechanic right now who's dying of a squamous cell carcinoma that arose from the back of his ear.

that the majority of skin cancers that we see that end up hurting people or killing patients don't arise in chronic sun-exposed areas to begin with. I have a 27-year-old patient right now dying of a metastatic melanoma that arose in completely sun protected skin. I have a 56-year-old mechanic right now who's dying of a squamous cell carcinoma that arose from the back of his ear.

And most experts who have this type of experience dealing with these patients have the same observation. So the question is, what's pulling the trigger? For the most part, UV plays a big role. the mechanism in which cancer forms, if we believe the basic high school biology is, hyperplasia, metaplasia, dysplasia, carcinoma.

And most experts who have this type of experience dealing with these patients have the same observation. So the question is, what's pulling the trigger? For the most part, UV plays a big role. the mechanism in which cancer forms, if we believe the basic high school biology is, hyperplasia, metaplasia, dysplasia, carcinoma.

And most experts who have this type of experience dealing with these patients have the same observation. So the question is, what's pulling the trigger? For the most part, UV plays a big role. the mechanism in which cancer forms, if we believe the basic high school biology is, hyperplasia, metaplasia, dysplasia, carcinoma.

So there's a trajectory of changes that are occurring as a result of cumulative mutations in the skin. If we believe that trajectory, then every skin cancer should see that change, and we just don't see that. Also, we know that skin cancers that arise in sun-damaged skin behave differently than the ones that are very lethal.