Dr. Teo Soleymani

That heterogeneous light, some have no improvement in psoriasis and some have great improvement. We were able to figure out which wavelengths make a big difference. It's around the 311, 312 nanometer range. sunlight does clear people's psoriasis up. That's why we see in northern latitudes where there's less sun intensity, the incidence of psoriasis is markedly higher than equatorial latitudes.

That heterogeneous light, some have no improvement in psoriasis and some have great improvement. We were able to figure out which wavelengths make a big difference. It's around the 311, 312 nanometer range. sunlight does clear people's psoriasis up. That's why we see in northern latitudes where there's less sun intensity, the incidence of psoriasis is markedly higher than equatorial latitudes.

And patients will say their psoriasis gets better when they're on a beach vacation. When they go back home, their psoriasis flares. There's also some component with vitamin D as well, but I think that's just secondary to the lack of UV exposure.

And patients will say their psoriasis gets better when they're on a beach vacation. When they go back home, their psoriasis flares. There's also some component with vitamin D as well, but I think that's just secondary to the lack of UV exposure.

And patients will say their psoriasis gets better when they're on a beach vacation. When they go back home, their psoriasis flares. There's also some component with vitamin D as well, but I think that's just secondary to the lack of UV exposure.

Yeah. So vitiligo is autoimmune, meaning your immune system is attacking itself. depigmentary skin disorder, meaning it's a condition in which your immune system attacks and kills the melanocytes, which are the cells that produce our skin's color. And we've found that it's an autoimmune condition from many basic science and elegant studies.

Yeah. So vitiligo is autoimmune, meaning your immune system is attacking itself. depigmentary skin disorder, meaning it's a condition in which your immune system attacks and kills the melanocytes, which are the cells that produce our skin's color. And we've found that it's an autoimmune condition from many basic science and elegant studies.

Yeah. So vitiligo is autoimmune, meaning your immune system is attacking itself. depigmentary skin disorder, meaning it's a condition in which your immune system attacks and kills the melanocytes, which are the cells that produce our skin's color. And we've found that it's an autoimmune condition from many basic science and elegant studies.

It's also very closely associated with other autoimmune conditions. So patients tend to also have atopic dermatitis, autoimmune thyroid disease, autoimmune anemia. There's some sort of immune dyscrasia that involves vitiligo patients. And what happens is the immune system at first paralyzes these cells with immune activity attacking them. And over time, these cells no longer survive and die off.

It's also very closely associated with other autoimmune conditions. So patients tend to also have atopic dermatitis, autoimmune thyroid disease, autoimmune anemia. There's some sort of immune dyscrasia that involves vitiligo patients. And what happens is the immune system at first paralyzes these cells with immune activity attacking them. And over time, these cells no longer survive and die off.

It's also very closely associated with other autoimmune conditions. So patients tend to also have atopic dermatitis, autoimmune thyroid disease, autoimmune anemia. There's some sort of immune dyscrasia that involves vitiligo patients. And what happens is the immune system at first paralyzes these cells with immune activity attacking them. And over time, these cells no longer survive and die off.

The cornerstone in treatment has always been some way to suppress the skin's immune response. Most of the time, the ligo tends to be focal, single areas, or segmental. Unfortunately, in certain cases, it can be whole body or completely depigmenting. Very uncommon, but we see this. We treat it by doing things that will help quiet the immune response in the skin.

The cornerstone in treatment has always been some way to suppress the skin's immune response. Most of the time, the ligo tends to be focal, single areas, or segmental. Unfortunately, in certain cases, it can be whole body or completely depigmenting. Very uncommon, but we see this. We treat it by doing things that will help quiet the immune response in the skin.

The cornerstone in treatment has always been some way to suppress the skin's immune response. Most of the time, the ligo tends to be focal, single areas, or segmental. Unfortunately, in certain cases, it can be whole body or completely depigmenting. Very uncommon, but we see this. We treat it by doing things that will help quiet the immune response in the skin.

So topical creams, topical steroids, et cetera, topical calcineurin inhibitors, which are non-steroidal anti-inflammatory medications. We also use certain wavelengths of UV light to treat this, eczema laser at 308 nanometers, UVB, UVA.

So topical creams, topical steroids, et cetera, topical calcineurin inhibitors, which are non-steroidal anti-inflammatory medications. We also use certain wavelengths of UV light to treat this, eczema laser at 308 nanometers, UVB, UVA.

So topical creams, topical steroids, et cetera, topical calcineurin inhibitors, which are non-steroidal anti-inflammatory medications. We also use certain wavelengths of UV light to treat this, eczema laser at 308 nanometers, UVB, UVA.

And then the renaissance in vitiligo has happened in the last three or four years with the identification of JAK inhibitors, which are these drugs that block the Janus kinase pathway. And we found that the JAK stat pathway plays a really important role in autoimmune mediation of melanocyte death.

And then the renaissance in vitiligo has happened in the last three or four years with the identification of JAK inhibitors, which are these drugs that block the Janus kinase pathway. And we found that the JAK stat pathway plays a really important role in autoimmune mediation of melanocyte death.

And then the renaissance in vitiligo has happened in the last three or four years with the identification of JAK inhibitors, which are these drugs that block the Janus kinase pathway. And we found that the JAK stat pathway plays a really important role in autoimmune mediation of melanocyte death.