Dr. Thomas Seyfried

No, no, I think that's pretty good. I think you did a good job on that. And I think that during this process of mitochondrial dysfunction that happens gradually, The mitochondria throw out what we call reactive oxygen species, ROS. These are radical species that can damage proteins, lipids, and the DNA, the nucleic acids, RNA and DNA, can be damaged by these ROS.

No, no, I think that's pretty good. I think you did a good job on that. And I think that during this process of mitochondrial dysfunction that happens gradually, The mitochondria throw out what we call reactive oxygen species, ROS. These are radical species that can damage proteins, lipids, and the DNA, the nucleic acids, RNA and DNA, can be damaged by these ROS.

No, no, I think that's pretty good. I think you did a good job on that. And I think that during this process of mitochondrial dysfunction that happens gradually, The mitochondria throw out what we call reactive oxygen species, ROS. These are radical species that can damage proteins, lipids, and the DNA, the nucleic acids, RNA and DNA, can be damaged by these ROS.

So they come out of the mitochondria, Because as the mitochondria of the cell becomes more and more dysfunctional, they produce, rather than producing ATP, the energy that they normally would produce, they produce ROS. And the ROS are carcinogenic and mutagenic. So the mutations that you see in the cancer cell are downstream effects of the damage to the reactive of the mitochondria.

So they come out of the mitochondria, Because as the mitochondria of the cell becomes more and more dysfunctional, they produce, rather than producing ATP, the energy that they normally would produce, they produce ROS. And the ROS are carcinogenic and mutagenic. So the mutations that you see in the cancer cell are downstream effects of the damage to the reactive of the mitochondria.

So they come out of the mitochondria, Because as the mitochondria of the cell becomes more and more dysfunctional, they produce, rather than producing ATP, the energy that they normally would produce, they produce ROS. And the ROS are carcinogenic and mutagenic. So the mutations that you see in the cancer cell are downstream effects of the damage to the reactive of the mitochondria.

So the field of cancer is focusing their energies and their attention on downstream stuff that is not relevant to the majority of cancers. So when you hear people speak of the ALK mutation, the P53 mutation and all these mutations, they're all downstream effects.

So the field of cancer is focusing their energies and their attention on downstream stuff that is not relevant to the majority of cancers. So when you hear people speak of the ALK mutation, the P53 mutation and all these mutations, they're all downstream effects.

So the field of cancer is focusing their energies and their attention on downstream stuff that is not relevant to the majority of cancers. So when you hear people speak of the ALK mutation, the P53 mutation and all these mutations, they're all downstream effects.

They're not the cause of cancer, they're the effects of the production of reactive oxygen species because the mitochondria have become abnormal. They're throwing out these radicals that are damaging lipids and proteins and causing mutations in the nucleus and the field, Almost the entire cancer field is focused on all this kind of stuff. And I'm saying, and others, and Warburg said, no, no, no.

They're not the cause of cancer, they're the effects of the production of reactive oxygen species because the mitochondria have become abnormal. They're throwing out these radicals that are damaging lipids and proteins and causing mutations in the nucleus and the field, Almost the entire cancer field is focused on all this kind of stuff. And I'm saying, and others, and Warburg said, no, no, no.

They're not the cause of cancer, they're the effects of the production of reactive oxygen species because the mitochondria have become abnormal. They're throwing out these radicals that are damaging lipids and proteins and causing mutations in the nucleus and the field, Almost the entire cancer field is focused on all this kind of stuff. And I'm saying, and others, and Warburg said, no, no, no.

You've got to go back and figure out where the energy is coming from. Because you're collecting mutations in the nucleus that are largely irrelevant, but they're coming because of the acidification in the microenvironment. They're coming because of the reactive oxygen species. But the most important question is what is the fuel that is driving the dysregulated cell growth? What's the energy?

You've got to go back and figure out where the energy is coming from. Because you're collecting mutations in the nucleus that are largely irrelevant, but they're coming because of the acidification in the microenvironment. They're coming because of the reactive oxygen species. But the most important question is what is the fuel that is driving the dysregulated cell growth? What's the energy?

You've got to go back and figure out where the energy is coming from. Because you're collecting mutations in the nucleus that are largely irrelevant, but they're coming because of the acidification in the microenvironment. They're coming because of the reactive oxygen species. But the most important question is what is the fuel that is driving the dysregulated cell growth? What's the energy?

What are these cells using to divide? And as you said, when we go back in time, we find out that all of the cells that existed on the planet were using those ancient fermentation pathways because there was no oxygen in the atmosphere. So they grew dysregulated growth. And the interesting thing is they would grow without regulation

What are these cells using to divide? And as you said, when we go back in time, we find out that all of the cells that existed on the planet were using those ancient fermentation pathways because there was no oxygen in the atmosphere. So they grew dysregulated growth. And the interesting thing is they would grow without regulation

What are these cells using to divide? And as you said, when we go back in time, we find out that all of the cells that existed on the planet were using those ancient fermentation pathways because there was no oxygen in the atmosphere. So they grew dysregulated growth. And the interesting thing is they would grow without regulation

until the fermentable fuels in the microenvironment were dissipated, and then these cells would up and die. So that told me right away that the way you kill cancer cells is you deprive them of their fermentable fuels. And because that's what's driving the dysregulated growth. They're dysregulated because the mitochondria are no longer in control of the system.

until the fermentable fuels in the microenvironment were dissipated, and then these cells would up and die. So that told me right away that the way you kill cancer cells is you deprive them of their fermentable fuels. And because that's what's driving the dysregulated growth. They're dysregulated because the mitochondria are no longer in control of the system.