Dr. Victor Carrión

They look at the genes that were changed, that their activity changed because of this cortisol exposure. And through epigenetic analysis, which is this space, you know, between DNA and RNA and there's like methylation patterns and all that. And some genes activity changes. Some turn on, some turn off.

So, interestingly, the majority of the genes that we found there were genes that have been addressed in the literature as potentially being related to post-traumatic stress disorder. Things like the glucocorticoid receptor genes and things that you would think of.

So, interestingly, the majority of the genes that we found there were genes that have been addressed in the literature as potentially being related to post-traumatic stress disorder. Things like the glucocorticoid receptor genes and things that you would think of.

So, interestingly, the majority of the genes that we found there were genes that have been addressed in the literature as potentially being related to post-traumatic stress disorder. Things like the glucocorticoid receptor genes and things that you would think of.

But there was another subset of genes that we identified that were novel, and I was very interested in those because of my interest in accelerated aging because of stress. And those were genes that are related to collagen formation. And we know that atherosclerosis has been related to stress, for example.

But there was another subset of genes that we identified that were novel, and I was very interested in those because of my interest in accelerated aging because of stress. And those were genes that are related to collagen formation. And we know that atherosclerosis has been related to stress, for example.

But there was another subset of genes that we identified that were novel, and I was very interested in those because of my interest in accelerated aging because of stress. And those were genes that are related to collagen formation. And we know that atherosclerosis has been related to stress, for example.

And we know that accelerated aging, not only in PTSD, but in mental health conditions overall, individuals that suffer from severe mental illness chronically in their life end up dying 25 years younger than the rest of the population. That's very significant. And so stress and accelerated aging, interesting. Okay, so these are interesting findings in organoids.

And we know that accelerated aging, not only in PTSD, but in mental health conditions overall, individuals that suffer from severe mental illness chronically in their life end up dying 25 years younger than the rest of the population. That's very significant. And so stress and accelerated aging, interesting. Okay, so these are interesting findings in organoids.

And we know that accelerated aging, not only in PTSD, but in mental health conditions overall, individuals that suffer from severe mental illness chronically in their life end up dying 25 years younger than the rest of the population. That's very significant. And so stress and accelerated aging, interesting. Okay, so these are interesting findings in organoids.

But when you have those, what you do is you move on to a population study. So these kids in Puerto Rico that are going through these interventions, besides me checking on their PTSD, their anxiety, their depression, they're giving me a vocal swab.

But when you have those, what you do is you move on to a population study. So these kids in Puerto Rico that are going through these interventions, besides me checking on their PTSD, their anxiety, their depression, they're giving me a vocal swab.

But when you have those, what you do is you move on to a population study. So these kids in Puerto Rico that are going through these interventions, besides me checking on their PTSD, their anxiety, their depression, they're giving me a vocal swab.

And in the vocal swab, those epithelial cells, we can actually take them through epigenetic analysis and see those kids at time one that, even though they've gone through all this trauma, may not be faring that much worse as their counterparts and compare them.

And in the vocal swab, those epithelial cells, we can actually take them through epigenetic analysis and see those kids at time one that, even though they've gone through all this trauma, may not be faring that much worse as their counterparts and compare them.

And in the vocal swab, those epithelial cells, we can actually take them through epigenetic analysis and see those kids at time one that, even though they've gone through all this trauma, may not be faring that much worse as their counterparts and compare them.

And not only that, we can actually also look at response, treatment response for the intervention, for the yoga and mindfulness preventive intervention, and for the treatment, for the Q Center therapy. So that's the plan. That's the plan in trying to bring more light into what is the biology of resilience and how can we understand resilience better.

And not only that, we can actually also look at response, treatment response for the intervention, for the yoga and mindfulness preventive intervention, and for the treatment, for the Q Center therapy. So that's the plan. That's the plan in trying to bring more light into what is the biology of resilience and how can we understand resilience better.

And not only that, we can actually also look at response, treatment response for the intervention, for the yoga and mindfulness preventive intervention, and for the treatment, for the Q Center therapy. So that's the plan. That's the plan in trying to bring more light into what is the biology of resilience and how can we understand resilience better.

And collaborations are key, right? Because the world is so complex now that there's no way that a single lab could have all this expertise. So you're right, a place like Stanford allows for these communications to happen, for these collaborations. to happen. In 28 years that I've been there, I have never heard, no, I'm not interested in that.