Eric Ravussin, Ph.D.

I don't have an answer to that because it's not my field, energy intake. But on the other hand, you just mentioned the GLP-1. I think these gut hormones, I mean, you take GLP-1, GIP, CCK, PYY. Glucagon. If you put glucagon in insulin, I was talking about the gut, the GI hormones.

I don't have an answer to that because it's not my field, energy intake. But on the other hand, you just mentioned the GLP-1. I think these gut hormones, I mean, you take GLP-1, GIP, CCK, PYY. Glucagon. If you put glucagon in insulin, I was talking about the gut, the GI hormones.

Anyway, I think that there is no question now we start to know more because they are such good targets for weight management. We start to know more about the physiology of these hormones. And I think that there is no question in my mind that the speed at which you deliver this food

Anyway, I think that there is no question now we start to know more because they are such good targets for weight management. We start to know more about the physiology of these hormones. And I think that there is no question in my mind that the speed at which you deliver this food

in the stomach and it leaves the stomach is very important for the kinetic of these gut hormones, which are important for the regulation of your food intake in general. I think this is one of the things that these GLP-1 and combination of peptides now has shed light on is that they are potent modulators of your intake.

in the stomach and it leaves the stomach is very important for the kinetic of these gut hormones, which are important for the regulation of your food intake in general. I think this is one of the things that these GLP-1 and combination of peptides now has shed light on is that they are potent modulators of your intake.

Now, the interaction between the exercise that you do before and the meal that you have 20 minutes after and the speed of the meal and all that, I don't know this triangle how it works, but I think it would be a very good topic of research to know the interaction between your physical activity or the bout of exercise, the speed at which you ingest the calories, and the delivery of these gut hormones.

Now, the interaction between the exercise that you do before and the meal that you have 20 minutes after and the speed of the meal and all that, I don't know this triangle how it works, but I think it would be a very good topic of research to know the interaction between your physical activity or the bout of exercise, the speed at which you ingest the calories, and the delivery of these gut hormones.

Yes, it was the question. Isocaloric is the important word.

Yes, it was the question. Isocaloric is the important word.

Because now, can you do that in the everyday life under not isocaloric condition, but what you want to eat?

Because now, can you do that in the everyday life under not isocaloric condition, but what you want to eat?

No, I think it was a question, and we were a little bit skeptical. It was the start of the carbohydrate-insulin model. We're used to the energy balance model, but I think it was a good question, and I like the way we debated that with the red team, the blue team, if I recall correctly, and we were arguing about the best design.

No, I think it was a question, and we were a little bit skeptical. It was the start of the carbohydrate-insulin model. We're used to the energy balance model, but I think it was a good question, and I like the way we debated that with the red team, the blue team, if I recall correctly, and we were arguing about the best design.

And at the end, now if I had to redesign this study, I would do it differently.

And at the end, now if I had to redesign this study, I would do it differently.

The experiment was done to do a isocaloric intake over four weeks, I think. We had the baseline diet, which was the SAD diet, the standard American diet, and we had after a low-carbohydrate diet or ketogenic diet for four weeks.

The experiment was done to do a isocaloric intake over four weeks, I think. We had the baseline diet, which was the SAD diet, the standard American diet, and we had after a low-carbohydrate diet or ketogenic diet for four weeks.

And the hypothesis was that, and this was led by the people who have been bringing up the carbohydrate-insulin model, saying that you create a basically uptake of these substrate from the blood into the storage, mostly the adipose tissue and maybe some in the liver.

And the hypothesis was that, and this was led by the people who have been bringing up the carbohydrate-insulin model, saying that you create a basically uptake of these substrate from the blood into the storage, mostly the adipose tissue and maybe some in the liver.