Sir John Bell

It's going to be, well, it's a bit of the T cells, but it's also a bit of the innate immune response.

And don't forget mate cells and don't forget a bit of this and that.

So we think that if we can assemble the right data set from these structured environments,

we can start to predict and anticipate which type of immune response you need to stimulate both for therapy and for protection against disease.

And hopefully that will actually create a whole scientific foundation for vaccine development, but also other kinds of immune therapy and things like cancer and potentially autoimmune disease as well.

So that's a big push.

For us, we're just busy.

The lab isn't set up.

We've got somebody to run the lab now.

We've got the human challenge model set up with Andy Pollard and colleagues.

So we're building that out.

And within six months, I think we'll be starting to collect data.

So I'm just kind of hoping we can get the immune system in a bit more structured.

Because you're absolutely right.

It's a bit pin the tail on the donkey at the moment.

You have no idea what's actually causing what.

No, it's amazing.

And, you know, you wouldn't have predicted a lot of this stuff.

And I...

I think that means that we haven't really got under the skin of the mechanistic events here, and we need to do more to try and get there.