A Breakthrough in Understanding Long COVID

People with long COVID experience persistent fatigue, breathlessness, and brain fog. Research links these effects to fibrin microclots intertwined with neutrophil extracellular traps that obstruct microvessels and impair oxygen delivery SARS-CoV-2 pushes blood into a hypercoagulable state, damaging endothelium, activating platelets, and inflammatory cytokines. This causes clot risk to persist for months, even after mild infections and hospital discharge Long COVID essentially boils down to mitochondrial dysfunction. Management includes proteolytic enzymes to help clear spike protein, while the I-RECOVER protocol guides detoxification, inflammation control, and mitochondrial repair to restore overall function Protecting cardiolipin, the mitochondrial membrane fat, requires limiting easily oxidized linoleic acid (LA) from seed oils, processed foods, nuts, seeds, and grain-fed meats Methylene blue may enhance mitochondrial respiration at low doses. Additionally, correcting copper deficiency and lowering excess iron through blood donation support energy production

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