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这篇文章详细介绍了小鼠和人类中风后脑部炎症的机制。具体来说，研究发现肥大细胞特异性受体 Mrgprb2/MRGPRX2 在中风后激活脑膜肥大细胞中起着关键作用。该激活导致头骨骨髓嗜中性粒细胞招募到脑中，加剧了炎症。作者们通过抑制 Mrgprb2/MRGPRX2 来减轻炎症并改善中风结果，从而确定了治疗靶点。该研究强调了脑膜肥大细胞在脑免疫中的核心作用以及头骨骨髓-脑膜-脑网络的复杂相互作用。References: Kothari R, Abdulrahim M W, Oh H J, et al. A mast cell receptor mediates post-stroke brain inflammation via a dural-brain axis[J]. Cell, 2025.
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