Description
这篇科学综述主要讨论了肥胖和2型糖尿病（T2D）这两种常见代谢紊乱的潜在原因和机制。文章重点阐述了胰岛素抵抗，将其描述为能量摄入与消耗失衡导致营养储存途径增加的结果。此外，该综述探讨了脂肪组织功能障碍、炎症途径、循环代谢物以及肝脏葡萄糖生成增加在T2D发病机制中的作用，并着重介绍了人类和相关动物模型的研究数据，旨在为T2D的未来精准医疗提供见解。References: Roden M, Shulman G I. The integrative biology of type 2 diabetes[J]. Nature, 2019, 576(7785): 51-60.
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