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欢迎收听今日医学播客！一项新研究揭示了喉癌治疗中的关键分子机制。研究发现，CTSL基因在喉癌组织中显著上调，并与不良预后相关。CTSL通过激活IL6-JAK-STAT3信号通路，促进喉癌细胞的增殖和自噬过程。基于CTSL的自噬调节作用，研究者开发了一种多基因签名，有望提高喉癌患者的临床预测能力。这一发现为喉癌的精准治疗提供了新思路。不要错过这一医学新突破！
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