欢迎收听医学前沿播客！科研喵使用AI读文献，祝你效率百倍，访问labcat.com.cn下载。最新研究发现，c-Met受体酪氨酸激酶（RTK）在心血管疾病中扮演重要角色，特别是在细胞增殖、存活、血管生成和组织修复方面。虽然c-Met在肿瘤学中已广泛研究，但其在心血管健康和疾病中的双重调节功能正逐渐被认识。在动脉粥样硬化中，c-Met具有促进血管平滑肌细胞增殖和炎症的矛盾效果，同时具有抗凋亡和抗纤维化的作用。预临床研究显示，c-Met在心肌梗死中具有心脏保护潜力，但异常激活可能加剧肺动脉高压。c-Met还影响心肌炎中的炎症细胞动态，并与神经激素途径相互作用调节心力衰竭的进展。针对c-Met的治疗策略在预临床模型中显示出潜力，但需要严格的临床验证。未来的研究必须阐明c-Met的时空信号机制，以利用其修复潜力，同时减轻病理效应，最终推进心血管疾病的精准治疗。
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