Dr. Elroy Vojdani

There are a large group of viruses that we as adults, by the time we're adults, we've been exposed to, we've been infected with. HHV-6, which is roseola, something that we typically get by the time we're three years old, not a big deal. If you are symptomatic, you've got a fever for a couple of days, you have rashes.

Epstein-Barr virus, the majority of adults are asymptomatic from the infection, same with CMV as well too. These viruses are genius in their long-term evolution against us. They have figured out how to evade complete immune eradication by hiding in tissue after the acute infection. But with a normal immune system, they stay in dormancy.

Epstein-Barr virus, the majority of adults are asymptomatic from the infection, same with CMV as well too. These viruses are genius in their long-term evolution against us. They have figured out how to evade complete immune eradication by hiding in tissue after the acute infection. But with a normal immune system, they stay in dormancy.

Epstein-Barr virus, the majority of adults are asymptomatic from the infection, same with CMV as well too. These viruses are genius in their long-term evolution against us. They have figured out how to evade complete immune eradication by hiding in tissue after the acute infection. But with a normal immune system, they stay in dormancy.

They wouldn't dare step out into the wild and get eradicated by the immune system. But what we're finding is that the best.

They wouldn't dare step out into the wild and get eradicated by the immune system. But what we're finding is that the best.

They wouldn't dare step out into the wild and get eradicated by the immune system. But what we're finding is that the best.

Correct. Right? It's not rolling around in the bloodstream active all day long. But a very, very large percentage of long COVID patients have viral reactivation as a core of their clinical symptoms and clinical disease. So again, that poses the question, what in the world is happening with the immune system in the short and long term following a COVID viral infection? It's not meeting

Correct. Right? It's not rolling around in the bloodstream active all day long. But a very, very large percentage of long COVID patients have viral reactivation as a core of their clinical symptoms and clinical disease. So again, that poses the question, what in the world is happening with the immune system in the short and long term following a COVID viral infection? It's not meeting

Correct. Right? It's not rolling around in the bloodstream active all day long. But a very, very large percentage of long COVID patients have viral reactivation as a core of their clinical symptoms and clinical disease. So again, that poses the question, what in the world is happening with the immune system in the short and long term following a COVID viral infection? It's not meeting

the demands in the short term and then not balancing itself in the long term as well, which provides a beautiful open window for these reactivated viruses.

the demands in the short term and then not balancing itself in the long term as well, which provides a beautiful open window for these reactivated viruses.

the demands in the short term and then not balancing itself in the long term as well, which provides a beautiful open window for these reactivated viruses.

So currently, with what we understand right now, I break it into five buckets. So there's viral persistence, which is essentially somebody never fully clears the initial COVID infection. They've got this very low level infection that just keeps on going and going and going and going. There's something called super antigen activation, which is parts of COVID have an ability to just dramatically

So currently, with what we understand right now, I break it into five buckets. So there's viral persistence, which is essentially somebody never fully clears the initial COVID infection. They've got this very low level infection that just keeps on going and going and going and going. There's something called super antigen activation, which is parts of COVID have an ability to just dramatically

So currently, with what we understand right now, I break it into five buckets. So there's viral persistence, which is essentially somebody never fully clears the initial COVID infection. They've got this very low level infection that just keeps on going and going and going and going. There's something called super antigen activation, which is parts of COVID have an ability to just dramatically

I'll just say piss off the immune system. There's the mitochondrial dysfunction and loss of autophagy that happens there. There's the microbiome and gut permeability dysfunction, and then there's the autoimmunity component. So if you're going to talk about diagnostics to be able to accurately pick up,

I'll just say piss off the immune system. There's the mitochondrial dysfunction and loss of autophagy that happens there. There's the microbiome and gut permeability dysfunction, and then there's the autoimmunity component. So if you're going to talk about diagnostics to be able to accurately pick up,

I'll just say piss off the immune system. There's the mitochondrial dysfunction and loss of autophagy that happens there. There's the microbiome and gut permeability dysfunction, and then there's the autoimmunity component. So if you're going to talk about diagnostics to be able to accurately pick up,

what's happening with long COVID, you basically have to say, okay, which one of these five buckets is the person living in? Everyone is gonna have some unique spectrum of those five, though the majority will have, let's say three or four of them. So we don't have diagnostics for the mitochondrial part, maybe on the research side.