Dr. Elroy Vojdani

what's happening with long COVID, you basically have to say, okay, which one of these five buckets is the person living in? Everyone is gonna have some unique spectrum of those five, though the majority will have, let's say three or four of them. So we don't have diagnostics for the mitochondrial part, maybe on the research side.

what's happening with long COVID, you basically have to say, okay, which one of these five buckets is the person living in? Everyone is gonna have some unique spectrum of those five, though the majority will have, let's say three or four of them. So we don't have diagnostics for the mitochondrial part, maybe on the research side.

They're real, for sure. The viral reactivation stuff, I think, you know, rather straightforward antibodies, IgM, IgG antibodies to different targets of Epstein-Barr virus, HHV-6, CMV. There's no diagnostics for COVID persistence, if that is in case what's going on.

They're real, for sure. The viral reactivation stuff, I think, you know, rather straightforward antibodies, IgM, IgG antibodies to different targets of Epstein-Barr virus, HHV-6, CMV. There's no diagnostics for COVID persistence, if that is in case what's going on.

They're real, for sure. The viral reactivation stuff, I think, you know, rather straightforward antibodies, IgM, IgG antibodies to different targets of Epstein-Barr virus, HHV-6, CMV. There's no diagnostics for COVID persistence, if that is in case what's going on.

I mean, you can look at whether there's very high levels of COVID antibody production for long periods of time, and you can infer that there's COVID persistence there. The autoimmune part of it. You brought up the lab in Germany that's doing an autoimmune panel specifically for long COVID.

I mean, you can look at whether there's very high levels of COVID antibody production for long periods of time, and you can infer that there's COVID persistence there. The autoimmune part of it. You brought up the lab in Germany that's doing an autoimmune panel specifically for long COVID.

I mean, you can look at whether there's very high levels of COVID antibody production for long periods of time, and you can infer that there's COVID persistence there. The autoimmune part of it. You brought up the lab in Germany that's doing an autoimmune panel specifically for long COVID.

In our studies as well, neurological targets like myelin basic protein, myelo-oligodendrocyte glycoprotein, the blood-brain barrier is a very common target that was demonstrated in mouse literature.

In our studies as well, neurological targets like myelin basic protein, myelo-oligodendrocyte glycoprotein, the blood-brain barrier is a very common target that was demonstrated in mouse literature.

In our studies as well, neurological targets like myelin basic protein, myelo-oligodendrocyte glycoprotein, the blood-brain barrier is a very common target that was demonstrated in mouse literature.

The most important defense of your brain, which is the blood-brain barrier, disrupted in football players, boxers, Alzheimer's, Parkinson's. So that same kind of core defense layer of the brain gets damaged by COVID. You can look at those markers in the blood. And then the specific neurological proteins like myelin basic protein, which is traditionally damaged in something like multiple sclerosis.

The most important defense of your brain, which is the blood-brain barrier, disrupted in football players, boxers, Alzheimer's, Parkinson's. So that same kind of core defense layer of the brain gets damaged by COVID. You can look at those markers in the blood. And then the specific neurological proteins like myelin basic protein, which is traditionally damaged in something like multiple sclerosis.

The most important defense of your brain, which is the blood-brain barrier, disrupted in football players, boxers, Alzheimer's, Parkinson's. So that same kind of core defense layer of the brain gets damaged by COVID. You can look at those markers in the blood. And then the specific neurological proteins like myelin basic protein, which is traditionally damaged in something like multiple sclerosis.

Yeah, make an attempt to try to, on this kind of early leading edge side of things, identify how much of each one of them they're dealing with.

Yeah, make an attempt to try to, on this kind of early leading edge side of things, identify how much of each one of them they're dealing with.

Yeah, make an attempt to try to, on this kind of early leading edge side of things, identify how much of each one of them they're dealing with.

It's tough because it's multiple pieces. But if I was going to break it down to what I think the core of it is, the acquired... mitochondrial damage and the associated lack of autophagy to me is really core there. So mitochondria are the powerhouse of the body. We know that for energy production, but I think it's underappreciated how much a damaged mitochondria will lead to

It's tough because it's multiple pieces. But if I was going to break it down to what I think the core of it is, the acquired... mitochondrial damage and the associated lack of autophagy to me is really core there. So mitochondria are the powerhouse of the body. We know that for energy production, but I think it's underappreciated how much a damaged mitochondria will lead to

It's tough because it's multiple pieces. But if I was going to break it down to what I think the core of it is, the acquired... mitochondrial damage and the associated lack of autophagy to me is really core there. So mitochondria are the powerhouse of the body. We know that for energy production, but I think it's underappreciated how much a damaged mitochondria will lead to