Dr. Eric Verdin

But interestingly, another anecdote, for example, someone did an interesting experiment where they measured the clocks at different times of the day in the same person. And they found over the 24-hour period, the clock would vary by five years.

But interestingly, another anecdote, for example, someone did an interesting experiment where they measured the clocks at different times of the day in the same person. And they found over the 24-hour period, the clock would vary by five years.

Meaning that if you're going to do this, do it every time at the same time of the day, because there's obviously a diurnal variation in there.

Meaning that if you're going to do this, do it every time at the same time of the day, because there's obviously a diurnal variation in there.

Meaning that if you're going to do this, do it every time at the same time of the day, because there's obviously a diurnal variation in there.

So maybe I'll just take it one step backwards and just to alert your audience to the fact that there will be no magic pill. Aging is an incredibly complex mechanism. And you alluded to earlier in our discussion to these hallmarks of aging, which is think about it as a catalog of all of these abnormalities that happen during aging.

So maybe I'll just take it one step backwards and just to alert your audience to the fact that there will be no magic pill. Aging is an incredibly complex mechanism. And you alluded to earlier in our discussion to these hallmarks of aging, which is think about it as a catalog of all of these abnormalities that happen during aging.

So maybe I'll just take it one step backwards and just to alert your audience to the fact that there will be no magic pill. Aging is an incredibly complex mechanism. And you alluded to earlier in our discussion to these hallmarks of aging, which is think about it as a catalog of all of these abnormalities that happen during aging.

And that includes epigenetic dysregulation, senescent cells, failed nutrient sensing, defective stem cells, defect in mitochondrial biogenesis. There's all kinds of problems that happens during aging. By the way, they're all connected to each other. And when we think about aging and solving it, I think if you study it, you will realize how complicated the process is.

And that includes epigenetic dysregulation, senescent cells, failed nutrient sensing, defective stem cells, defect in mitochondrial biogenesis. There's all kinds of problems that happens during aging. By the way, they're all connected to each other. And when we think about aging and solving it, I think if you study it, you will realize how complicated the process is.

And that includes epigenetic dysregulation, senescent cells, failed nutrient sensing, defective stem cells, defect in mitochondrial biogenesis. There's all kinds of problems that happens during aging. By the way, they're all connected to each other. And when we think about aging and solving it, I think if you study it, you will realize how complicated the process is.

And that, you know, we at the Buck Institute, we have about 300 people working on aging. We don't take sides in a way that I cannot predict what's going to be more important. So we study pretty much every one of the hallmarks of aging. We obviously have a big emphasis on senescence because it was mostly discovered through the work of Jeanne Campisi at the back.

And that, you know, we at the Buck Institute, we have about 300 people working on aging. We don't take sides in a way that I cannot predict what's going to be more important. So we study pretty much every one of the hallmarks of aging. We obviously have a big emphasis on senescence because it was mostly discovered through the work of Jeanne Campisi at the back.

And that, you know, we at the Buck Institute, we have about 300 people working on aging. We don't take sides in a way that I cannot predict what's going to be more important. So we study pretty much every one of the hallmarks of aging. We obviously have a big emphasis on senescence because it was mostly discovered through the work of Jeanne Campisi at the back.

So many of us are still working on senescence, but we have people working on mitochondria. We have people working on stem cells with people working on metabolism and so on. So I think one of the things that is really emerging right now is the complexity.

So many of us are still working on senescence, but we have people working on mitochondria. We have people working on stem cells with people working on metabolism and so on. So I think one of the things that is really emerging right now is the complexity.

So many of us are still working on senescence, but we have people working on mitochondria. We have people working on stem cells with people working on metabolism and so on. So I think one of the things that is really emerging right now is the complexity.

It's important because, you know, frankly, I have... I've sometimes been called the grumpy man of longevity research because I refuse the hype or work. I think there's a lot of really exciting things happening in aging research, but I'm not deluding myself. I don't tell people I'm going to live to 150 or 170 like some of my colleagues do.

It's important because, you know, frankly, I have... I've sometimes been called the grumpy man of longevity research because I refuse the hype or work. I think there's a lot of really exciting things happening in aging research, but I'm not deluding myself. I don't tell people I'm going to live to 150 or 170 like some of my colleagues do.

It's important because, you know, frankly, I have... I've sometimes been called the grumpy man of longevity research because I refuse the hype or work. I think there's a lot of really exciting things happening in aging research, but I'm not deluding myself. I don't tell people I'm going to live to 150 or 170 like some of my colleagues do.