Dr. Mark Gordon

Absolutely. So I take 500 milligrams twice a day.

And how much zinc with that? 30 milligrams. Okay. I take 30. And you do that twice a day? I do the quercetin twice a day, but zinc, because my levels are where they're at, I don't put a lot of zinc in because zinc's involved in about 300 processes in the body. It's antiviral that we just talked about. It's anti-Alzheimer's because it turns out

And how much zinc with that? 30 milligrams. Okay. I take 30. And you do that twice a day? I do the quercetin twice a day, but zinc, because my levels are where they're at, I don't put a lot of zinc in because zinc's involved in about 300 processes in the body. It's antiviral that we just talked about. It's anti-Alzheimer's because it turns out

And how much zinc with that? 30 milligrams. Okay. I take 30. And you do that twice a day? I do the quercetin twice a day, but zinc, because my levels are where they're at, I don't put a lot of zinc in because zinc's involved in about 300 processes in the body. It's antiviral that we just talked about. It's anti-Alzheimer's because it turns out

that the production of the chemical called beta amyloid, there's an enzyme that regulates it, and it's zinc-dependent. So if it's working, it's called secretase. It's called alpha secretase. It's zinc-dependent. Beta secretase is not. So beta secretase takes and makes the beta amyloid that causes the Alzheimer's, the inflammation.

that the production of the chemical called beta amyloid, there's an enzyme that regulates it, and it's zinc-dependent. So if it's working, it's called secretase. It's called alpha secretase. It's zinc-dependent. Beta secretase is not. So beta secretase takes and makes the beta amyloid that causes the Alzheimer's, the inflammation.

that the production of the chemical called beta amyloid, there's an enzyme that regulates it, and it's zinc-dependent. So if it's working, it's called secretase. It's called alpha secretase. It's zinc-dependent. Beta secretase is not. So beta secretase takes and makes the beta amyloid that causes the Alzheimer's, the inflammation.

And with that inflammation, you then start getting the same thing in CTE. So in all these inflammatory conditions, they have the same beta amyloid and cause for CTE, the hyperphospholated tau protein that we call NFTs, neurofibrotangles. Okay. So they're all related.

And with that inflammation, you then start getting the same thing in CTE. So in all these inflammatory conditions, they have the same beta amyloid and cause for CTE, the hyperphospholated tau protein that we call NFTs, neurofibrotangles. Okay. So they're all related.

And with that inflammation, you then start getting the same thing in CTE. So in all these inflammatory conditions, they have the same beta amyloid and cause for CTE, the hyperphospholated tau protein that we call NFTs, neurofibrotangles. Okay. So they're all related.

So what quercetin does is it increases mitochondrial replication in about seven days, doubles the amount of mitochondria intracellularly. It helps increase and deliver something called IGF binding protein 3, insulin-like binding protein 3. Binding protein 3 is always looked at as being the carrier for IGF-1, insulin-like growth factor, growth hormone.

So what quercetin does is it increases mitochondrial replication in about seven days, doubles the amount of mitochondria intracellularly. It helps increase and deliver something called IGF binding protein 3, insulin-like binding protein 3. Binding protein 3 is always looked at as being the carrier for IGF-1, insulin-like growth factor, growth hormone.

So what quercetin does is it increases mitochondrial replication in about seven days, doubles the amount of mitochondria intracellularly. It helps increase and deliver something called IGF binding protein 3, insulin-like binding protein 3. Binding protein 3 is always looked at as being the carrier for IGF-1, insulin-like growth factor, growth hormone.

Turns on in the liver the production of insulin-like growth factor, which is the main below-the-neck growth factor for our body. Improves protein synthesis. Decreases inflammation, too.

Turns on in the liver the production of insulin-like growth factor, which is the main below-the-neck growth factor for our body. Improves protein synthesis. Decreases inflammation, too.

Turns on in the liver the production of insulin-like growth factor, which is the main below-the-neck growth factor for our body. Improves protein synthesis. Decreases inflammation, too.

Well in the train of thought on Alzheimer's, you know, they're saying that it's due to the recessive genes Well, if you look at the real studies recently 95% of the cases of Alzheimer's disease appear to be due to trauma and aging and Trauma and aging. Only 5%. So trauma, like head trauma? Head trauma.

Well in the train of thought on Alzheimer's, you know, they're saying that it's due to the recessive genes Well, if you look at the real studies recently 95% of the cases of Alzheimer's disease appear to be due to trauma and aging and Trauma and aging. Only 5%. So trauma, like head trauma? Head trauma.

Well in the train of thought on Alzheimer's, you know, they're saying that it's due to the recessive genes Well, if you look at the real studies recently 95% of the cases of Alzheimer's disease appear to be due to trauma and aging and Trauma and aging. Only 5%. So trauma, like head trauma? Head trauma.

Because what happens is trauma stimulates the brain because of inflammation to increase the production of beta amyloid. And it's because they found recently another secretase. What secretases are are the enzymes that convert a protein called APP, Alzheimer's precursor protein. And it's a long protein. And two enzymes go in and clip it here and clip it here. And that piece is beta amyloid.