Dr. Michael Snyder

And I'm happy to talk about that, but that's still in progress.

So I wouldn't say we've totally done what you've gotten at, but we would like to dissect the resistance training versus an aerobic or endurance type of training.

I mean, the bottom line is, of course, exercising any time is better than not exercising at all.

So I think we'd all agree with that.

But we do think you get better glucose benefits if you are muscle and resistant during the morning.

And I also do believe that, yeah, building your muscle mass will help with actually reducing muscle and resistance.

But it's more than that, meaning there's a beta cell defect where you don't release insulin from your pancreas.

That has nothing to do with insulin resistance.

That's more a mechanistic thing.

Now, why that defect exists isn't so clear.

My case is kind of interesting, although we still don't fully understand it.

But then there's also incretin defects.

So incretin are these GLPs that everybody's heard about.

Ozempic is a mimic of those and Mongero and things like that.

And so there are people with defects that way.

So we're all different, and we can now subtype that.

We can say this person's got mostly an incretin defect.

This one's muslin-resistant.

This is a beta cell defect.

And some people are combinations of those.