Dr. Stephanie Venn-Watson

That's right.

That's right. That's right. Thank you for that. And your book, by the way, does a great job explaining all of those. It's hard to explain, trust me. If anyone wants to dive in more, please read your book, Young Forever. So then the second one exactly feeds into the hallmarks of aging. There's 12. The list keeps getting longer, it feels like, every couple of years.

That's right. That's right. Thank you for that. And your book, by the way, does a great job explaining all of those. It's hard to explain, trust me. If anyone wants to dive in more, please read your book, Young Forever. So then the second one exactly feeds into the hallmarks of aging. There's 12. The list keeps getting longer, it feels like, every couple of years.

That's right. That's right. Thank you for that. And your book, by the way, does a great job explaining all of those. It's hard to explain, trust me. If anyone wants to dive in more, please read your book, Young Forever. So then the second one exactly feeds into the hallmarks of aging. There's 12. The list keeps getting longer, it feels like, every couple of years.

Yeah, so there's now multiple studies showing that C15 targets six of them. And that includes helping C15 directly repairs mitochondrial function. It has a skip function where it's able to help restore broken mitochondria by enabling it to make energy. when normally it wouldn't be able to, which is exciting and important. It helps with poor cellular signaling.

Yeah, so there's now multiple studies showing that C15 targets six of them. And that includes helping C15 directly repairs mitochondrial function. It has a skip function where it's able to help restore broken mitochondria by enabling it to make energy. when normally it wouldn't be able to, which is exciting and important. It helps with poor cellular signaling.

Yeah, so there's now multiple studies showing that C15 targets six of them. And that includes helping C15 directly repairs mitochondrial function. It has a skip function where it's able to help restore broken mitochondria by enabling it to make energy. when normally it wouldn't be able to, which is exciting and important. It helps with poor cellular signaling.

We know that it not only activates AMPK and inhibits mTOR, but it also activates PPAR alpha delta and AKT, and it inhibits JAK-STAT and HDAC6. So that was a bunch of letters.

We know that it not only activates AMPK and inhibits mTOR, but it also activates PPAR alpha delta and AKT, and it inhibits JAK-STAT and HDAC6. So that was a bunch of letters.

We know that it not only activates AMPK and inhibits mTOR, but it also activates PPAR alpha delta and AKT, and it inhibits JAK-STAT and HDAC6. So that was a bunch of letters.

Right. So with the dolphins, it was pretty, it ended up being pretty straightforward. So they have an option of five different fish types and or they might be fed different fish types based upon what their job is at the Navy.

Right. So with the dolphins, it was pretty, it ended up being pretty straightforward. So they have an option of five different fish types and or they might be fed different fish types based upon what their job is at the Navy.

Right. So with the dolphins, it was pretty, it ended up being pretty straightforward. So they have an option of five different fish types and or they might be fed different fish types based upon what their job is at the Navy.

So if they're doing a job in which they're going through, you know, training of learning how to find an object, for example, they might get a bunch of low fat fish that they get kind of given throughout the whole day, it ends up that those low-fat fish had no C15 in them.

So if they're doing a job in which they're going through, you know, training of learning how to find an object, for example, they might get a bunch of low fat fish that they get kind of given throughout the whole day, it ends up that those low-fat fish had no C15 in them.

So if they're doing a job in which they're going through, you know, training of learning how to find an object, for example, they might get a bunch of low fat fish that they get kind of given throughout the whole day, it ends up that those low-fat fish had no C15 in them.

And what we were able to get to, remarkably, because the Navy's vigilant data collection, we were able to see that some dolphins were getting a higher C15 diet than other dolphins. And then even better, we did two Navy-funded studies in which we gave those dolphins with lower C15 a higher C15 fish diet, and we saw that their aging-related conditions reverse.

And what we were able to get to, remarkably, because the Navy's vigilant data collection, we were able to see that some dolphins were getting a higher C15 diet than other dolphins. And then even better, we did two Navy-funded studies in which we gave those dolphins with lower C15 a higher C15 fish diet, and we saw that their aging-related conditions reverse.

And what we were able to get to, remarkably, because the Navy's vigilant data collection, we were able to see that some dolphins were getting a higher C15 diet than other dolphins. And then even better, we did two Navy-funded studies in which we gave those dolphins with lower C15 a higher C15 fish diet, and we saw that their aging-related conditions reverse.

So then we were like, oh, okay, now we're onto something.