Dr. Stephanie Venn-Watson

Two thirds. So if you have fatty liver disease, you had an even higher risk of what we were saying the general population has.

Yeah, exactly. And then for the people who took a C15 supplementation and got their blood levels above that threshold of deficiency, they had lower liver enzymes, lower ALT, lower AST, and improved red blood cell health within 12 weeks. So- Now, the leading hypothesis and what we have found in the dolphins is that nutritional C15 deficiencies may actually

Yeah, exactly. And then for the people who took a C15 supplementation and got their blood levels above that threshold of deficiency, they had lower liver enzymes, lower ALT, lower AST, and improved red blood cell health within 12 weeks. So- Now, the leading hypothesis and what we have found in the dolphins is that nutritional C15 deficiencies may actually

Yeah, exactly. And then for the people who took a C15 supplementation and got their blood levels above that threshold of deficiency, they had lower liver enzymes, lower ALT, lower AST, and improved red blood cell health within 12 weeks. So- Now, the leading hypothesis and what we have found in the dolphins is that nutritional C15 deficiencies may actually

be the cause or at least one cause of a substantial phenotype of fatty liver? Because why did it show up? Why? Like why did this show up all of a sudden? In fact, so many. So that is obviously raising the urgency.

be the cause or at least one cause of a substantial phenotype of fatty liver? Because why did it show up? Why? Like why did this show up all of a sudden? In fact, so many. So that is obviously raising the urgency.

be the cause or at least one cause of a substantial phenotype of fatty liver? Because why did it show up? Why? Like why did this show up all of a sudden? In fact, so many. So that is obviously raising the urgency.

Yeah, and for us, we keep going back to the dolphins. And for the dolphin, we call this the dolphin phenotype of fatty liver disease, the only change they had, they had moved to, they were eating a high fish, high fat fish called the eulachon. which is called a candlefish. It has so much fat in it that you can stick a wick in it. You can dry it, stick a wick in it, and be a candle.

Yeah, and for us, we keep going back to the dolphins. And for the dolphin, we call this the dolphin phenotype of fatty liver disease, the only change they had, they had moved to, they were eating a high fish, high fat fish called the eulachon. which is called a candlefish. It has so much fat in it that you can stick a wick in it. You can dry it, stick a wick in it, and be a candle.

Yeah, and for us, we keep going back to the dolphins. And for the dolphin, we call this the dolphin phenotype of fatty liver disease, the only change they had, they had moved to, they were eating a high fish, high fat fish called the eulachon. which is called a candlefish. It has so much fat in it that you can stick a wick in it. You can dry it, stick a wick in it, and be a candle.

That fishery died out in the 1990s. And so that's when they moved to this lower-fat capelin And so at the same time that humans, we were moving away from whole fat dairy in our C15 sources, the dolphins were moving, going on a low C15 diet, and fatty liver disease pops up.

That fishery died out in the 1990s. And so that's when they moved to this lower-fat capelin And so at the same time that humans, we were moving away from whole fat dairy in our C15 sources, the dolphins were moving, going on a low C15 diet, and fatty liver disease pops up.

That fishery died out in the 1990s. And so that's when they moved to this lower-fat capelin And so at the same time that humans, we were moving away from whole fat dairy in our C15 sources, the dolphins were moving, going on a low C15 diet, and fatty liver disease pops up.

Yeah, and now we understand even more so how valuable that was.

Yeah, and now we understand even more so how valuable that was.

Yeah, and now we understand even more so how valuable that was.

that kind of as far as we also know that that's a human trial that seems to be that's right yeah and so that was published directionally showed what's possible like it didn't cure their fatty liver disease but it seems to sort of create a downward trend in the effects and you still have to cut out the sugar and starch yeah we need to look at the whole picture of lifestyles and how we're changing things what's interesting is it really goes against the whole like

that kind of as far as we also know that that's a human trial that seems to be that's right yeah and so that was published directionally showed what's possible like it didn't cure their fatty liver disease but it seems to sort of create a downward trend in the effects and you still have to cut out the sugar and starch yeah we need to look at the whole picture of lifestyles and how we're changing things what's interesting is it really goes against the whole like

that kind of as far as we also know that that's a human trial that seems to be that's right yeah and so that was published directionally showed what's possible like it didn't cure their fatty liver disease but it seems to sort of create a downward trend in the effects and you still have to cut out the sugar and starch yeah we need to look at the whole picture of lifestyles and how we're changing things what's interesting is it really goes against the whole like

idea of um all saturated fats like are bad and that we have one that's essential so it's like how do we get it back into our diet um so there's you know a lot of uh you could feel right the urgency around this movement um the reason for the book um is really to help increase education and let's have the conversations around so we can't explain it away anymore so you know