Dr. Yousef Elyaman

Yeah, their blood sugar normal.

Correct? It's the huge driver of fatty liver disease. I would say it's the number one driver, but there are eight or nine drivers. So what happens is that due to inflammation, due to toxicity, due to too much sugar, like too much of a sugar overload or a carbohydrate overload,

Correct? It's the huge driver of fatty liver disease. I would say it's the number one driver, but there are eight or nine drivers. So what happens is that due to inflammation, due to toxicity, due to too much sugar, like too much of a sugar overload or a carbohydrate overload,

Correct? It's the huge driver of fatty liver disease. I would say it's the number one driver, but there are eight or nine drivers. So what happens is that due to inflammation, due to toxicity, due to too much sugar, like too much of a sugar overload or a carbohydrate overload,

when those receptors on our fat cells, we have these insulin receptors that are supposed to take sugar and put sugar into the cells. When they start to not work well, when they start to get resistant, there is an enzyme called hormone sensitive lipase. And what that enzyme does is it starts tearing up the fat in the fat cell and it sends the fat to the liver and it overloads the liver.

when those receptors on our fat cells, we have these insulin receptors that are supposed to take sugar and put sugar into the cells. When they start to not work well, when they start to get resistant, there is an enzyme called hormone sensitive lipase. And what that enzyme does is it starts tearing up the fat in the fat cell and it sends the fat to the liver and it overloads the liver.

when those receptors on our fat cells, we have these insulin receptors that are supposed to take sugar and put sugar into the cells. When they start to not work well, when they start to get resistant, there is an enzyme called hormone sensitive lipase. And what that enzyme does is it starts tearing up the fat in the fat cell and it sends the fat to the liver and it overloads the liver.

So now you have this liver that gets overloaded. So the liver ends up with a whole bunch of triglycerides, which is one of the tests that we look on the lipid panel. Now, here's the interesting thing. So you could check a triglyceride level, and their level would look okay. And that's because some people genetically will decrease triglycerides in the blood. Increase it a lot.

So now you have this liver that gets overloaded. So the liver ends up with a whole bunch of triglycerides, which is one of the tests that we look on the lipid panel. Now, here's the interesting thing. So you could check a triglyceride level, and their level would look okay. And that's because some people genetically will decrease triglycerides in the blood. Increase it a lot.

So now you have this liver that gets overloaded. So the liver ends up with a whole bunch of triglycerides, which is one of the tests that we look on the lipid panel. Now, here's the interesting thing. So you could check a triglyceride level, and their level would look okay. And that's because some people genetically will decrease triglycerides in the blood. Increase it a lot.

What the triglycerides do, when you end up sending a whole bunch of triglycerides to the liver, they turn into something called VLDL. Very low density lipoprotein. Triglyceride rich VLDL versus triglyceride poor. And VLDL becomes, there's another enzyme found in the lining of our blood vessel that is called lipoprotein lipase. And it turns it into the tiny dense LDL.

What the triglycerides do, when you end up sending a whole bunch of triglycerides to the liver, they turn into something called VLDL. Very low density lipoprotein. Triglyceride rich VLDL versus triglyceride poor. And VLDL becomes, there's another enzyme found in the lining of our blood vessel that is called lipoprotein lipase. And it turns it into the tiny dense LDL.

What the triglycerides do, when you end up sending a whole bunch of triglycerides to the liver, they turn into something called VLDL. Very low density lipoprotein. Triglyceride rich VLDL versus triglyceride poor. And VLDL becomes, there's another enzyme found in the lining of our blood vessel that is called lipoprotein lipase. And it turns it into the tiny dense LDL.

which causes clogging of the blood vessel and heart disease, strokes, and heart attacks.

which causes clogging of the blood vessel and heart disease, strokes, and heart attacks.

which causes clogging of the blood vessel and heart disease, strokes, and heart attacks.

Right, yeah. What's happening in the liver? So what's happening in the liver? So we can go back to what's happening in the fat and then go right back to the liver. So number one, the fat starts to send the triglycerides to the liver and start overloading the liver.

Right, yeah. What's happening in the liver? So what's happening in the liver? So we can go back to what's happening in the fat and then go right back to the liver. So number one, the fat starts to send the triglycerides to the liver and start overloading the liver.

Right, yeah. What's happening in the liver? So what's happening in the liver? So we can go back to what's happening in the fat and then go right back to the liver. So number one, the fat starts to send the triglycerides to the liver and start overloading the liver.

When that process is overloaded, then your fat cells, which actually your fat is, I don't know if you've done a podcast on this, but your fat is like an endocrine organ. It makes things like adiponectin. When the process is bad, adiponectin levels can go down, which causes more insulin resistance and more inflammation.