Max Lugavere

Um, and even metabolic indicators and, and blood biomarkers, like we're now starting to, you know, correlate levels of amyloid in the blood to, um, amyloid in the brain.

So it's there, it's definitely involved and especially late in the game, um, in, in Alzheimer's disease.

I mean, it does start to build and strangle neurons essentially because it builds up in the space around neurons and neurons are, you know, I mean, there are brain cells that are like, how are,

You know, they make up the networks of, you know, cellular machinery that are responsible for our thinking and feeling and like everything that it makes means to be human, right?

But what's actually causing it to aggregate in the brain?

That is the looming question.

And so...

Yeah, I think that the way that the field is turning, it's no longer looking at amyloid as being causal.

We're trying to get ahead of that.

And I think we're succeeding in many ways.

I mean, it could have something to do with insulin sensitivity in the body.

We know that insulin resistance in the body is strongly inversely correlated to glucose metabolism in the brain.

So you want to make sure that you're insulin sensitive.

You want to

As I mentioned, you want to not be a type two diabetic.

We know clearly that there's a vascular component to Alzheimer's disease.

So you want to make sure that you don't have hypertension.

That's very important.

You don't want to be obese.

Obesity increases your risk for hypertension.