Nick Norwitz

Well, it's important to know what's the absolute risk because what you might be trading by reintroducing carbs is an increased risk of flares and ending up in the hospital or trying a medication that might have side effects, promote insulin resistance or other negative health effects that you don't otherwise want. So I'm not here to say that LZL or Applebee is irrelevant.

I'm here to say it's context dependent and that in assessing an individual's risk, you need to take a look at the whole picture. So, you know, TLDR is going over your head. Don't get your medical information, your health recommendations off of podcasts and YouTube. I'm here to provide information. But at the end of the day, key thing is to assess your own risk profile.

I'm here to say it's context dependent and that in assessing an individual's risk, you need to take a look at the whole picture. So, you know, TLDR is going over your head. Don't get your medical information, your health recommendations off of podcasts and YouTube. I'm here to provide information. But at the end of the day, key thing is to assess your own risk profile.

I'm here to say it's context dependent and that in assessing an individual's risk, you need to take a look at the whole picture. So, you know, TLDR is going over your head. Don't get your medical information, your health recommendations off of podcasts and YouTube. I'm here to provide information. But at the end of the day, key thing is to assess your own risk profile.

But with that caveat in place. A question that I think we should ask is why does LDL go up? We talk all the time about LDL, but not so much about the physiology behind what drives it. And people then tend to oversimplify and think, oh, it's just saturated fat. They're just, quote, butter guzzlers. Or it's just some quirk of genetics, right? Those are kind of hand wavy statements.

But with that caveat in place. A question that I think we should ask is why does LDL go up? We talk all the time about LDL, but not so much about the physiology behind what drives it. And people then tend to oversimplify and think, oh, it's just saturated fat. They're just, quote, butter guzzlers. Or it's just some quirk of genetics, right? Those are kind of hand wavy statements.

But with that caveat in place. A question that I think we should ask is why does LDL go up? We talk all the time about LDL, but not so much about the physiology behind what drives it. And people then tend to oversimplify and think, oh, it's just saturated fat. They're just, quote, butter guzzlers. Or it's just some quirk of genetics, right? Those are kind of hand wavy statements.

And in some cases, they're true. Saturated fat can increase LDL, at least a little bit. Genetics can play a role. But when you have a really interesting phenomenon, like someone going low carbon, their LDL going for like 300, 400, 500, saturated fat almost certainly isn't the only thing at play. And in some people, it's very weird because they even go like plant-based low carbon.

And in some cases, they're true. Saturated fat can increase LDL, at least a little bit. Genetics can play a role. But when you have a really interesting phenomenon, like someone going low carbon, their LDL going for like 300, 400, 500, saturated fat almost certainly isn't the only thing at play. And in some people, it's very weird because they even go like plant-based low carbon.

And in some cases, they're true. Saturated fat can increase LDL, at least a little bit. Genetics can play a role. But when you have a really interesting phenomenon, like someone going low carbon, their LDL going for like 300, 400, 500, saturated fat almost certainly isn't the only thing at play. And in some people, it's very weird because they even go like plant-based low carbon.

They'll still see this response. I can actually get to my LDL for like 300, 400 on a vegan keto diet. So you get to ask the question, well, what's going on here and what can we learn from it? That's where this population that we've been studying comes into play.

They'll still see this response. I can actually get to my LDL for like 300, 400 on a vegan keto diet. So you get to ask the question, well, what's going on here and what can we learn from it? That's where this population that we've been studying comes into play.

They'll still see this response. I can actually get to my LDL for like 300, 400 on a vegan keto diet. So you get to ask the question, well, what's going on here and what can we learn from it? That's where this population that we've been studying comes into play.

Because what appears to happen when lean insulin sensitive people go low carb is when they're no longer relying on carbs as much for fuel, specifically what happens is the carb stores in the liver, which defend blood glucose stores when they drop, it triggers this cycle whereby you're burning fat

Because what appears to happen when lean insulin sensitive people go low carb is when they're no longer relying on carbs as much for fuel, specifically what happens is the carb stores in the liver, which defend blood glucose stores when they drop, it triggers this cycle whereby you're burning fat

Because what appears to happen when lean insulin sensitive people go low carb is when they're no longer relying on carbs as much for fuel, specifically what happens is the carb stores in the liver, which defend blood glucose stores when they drop, it triggers this cycle whereby you're burning fat

And some of the fat that kind of, you could say, spills over gets taken back up into the liver and then recycled through the body through a trafficking system that depends on cholesterol. So specifically, you have these big, you could say, fat-carrying boats. They're called VLDL particles, very low-density lipid particles.

And some of the fat that kind of, you could say, spills over gets taken back up into the liver and then recycled through the body through a trafficking system that depends on cholesterol. So specifically, you have these big, you could say, fat-carrying boats. They're called VLDL particles, very low-density lipid particles.

And some of the fat that kind of, you could say, spills over gets taken back up into the liver and then recycled through the body through a trafficking system that depends on cholesterol. So specifically, you have these big, you could say, fat-carrying boats. They're called VLDL particles, very low-density lipid particles.

You need them because fat, stored fat, doesn't mix with aqueous solution, so water. You know that if you take water, Oil, you put it in water, it doesn't mix. So it's the same way with fat in your body. It doesn't really mix well with blood. So you need a way to carry it around the body. And it gets packaged, the fat, into these shipment containers that contain cholesterol.