Paul Saladino, MD

And as you know, with a meta analysis, a researcher like Darius Mazzafari and at Tufts who gifted us with the food compass system, which tells us that Fruit Loops and Cheerios are healthier than ground beef and eggs. can write a meta-analysis on seed oils, and he's done this, and he can leave out things like Sydney Diet Heart.

He can just choose which trials he wants to put in his meta-analysis and write in the abstract that seed oils are benign or trend toward cardiovascular benefit for humans. Wow. This is ludicrous. This is ludicrous. This is the state of the affairs right now.

He can just choose which trials he wants to put in his meta-analysis and write in the abstract that seed oils are benign or trend toward cardiovascular benefit for humans. Wow. This is ludicrous. This is ludicrous. This is the state of the affairs right now.

He can just choose which trials he wants to put in his meta-analysis and write in the abstract that seed oils are benign or trend toward cardiovascular benefit for humans. Wow. This is ludicrous. This is ludicrous. This is the state of the affairs right now.

I hope it changes soon.

I hope it changes soon.

I hope it changes soon.

No. Native LDL. Native LDL. you would have a hard time arguing, looking at the medical literature, that native LDL, that is unoxidized, unmodified LDL is atherogenic, right? So something has to happen to our LDL to make it participate. Either to the LDL or to the endothelial wall or both.

No. Native LDL. Native LDL. you would have a hard time arguing, looking at the medical literature, that native LDL, that is unoxidized, unmodified LDL is atherogenic, right? So something has to happen to our LDL to make it participate. Either to the LDL or to the endothelial wall or both.

No. Native LDL. Native LDL. you would have a hard time arguing, looking at the medical literature, that native LDL, that is unoxidized, unmodified LDL is atherogenic, right? So something has to happen to our LDL to make it participate. Either to the LDL or to the endothelial wall or both.

I would argue the endothelial wall is the proximate event of atherosclerosis because we know that really uninjured endothelium, uninjured arteries don't really accumulate So you can think about this, and we may have talked about this on a previous podcast, but if we're talking about LDL or ApoB containing lipoproteins, which is a slightly broader family than just LDL,

I would argue the endothelial wall is the proximate event of atherosclerosis because we know that really uninjured endothelium, uninjured arteries don't really accumulate So you can think about this, and we may have talked about this on a previous podcast, but if we're talking about LDL or ApoB containing lipoproteins, which is a slightly broader family than just LDL,

I would argue the endothelial wall is the proximate event of atherosclerosis because we know that really uninjured endothelium, uninjured arteries don't really accumulate So you can think about this, and we may have talked about this on a previous podcast, but if we're talking about LDL or ApoB containing lipoproteins, which is a slightly broader family than just LDL,

We have, I have the same amount of LDL circulating in my veins. You know, you have veins on your arm too. You can see these are returning blood to my heart from my hands. Deeper in my arm, I have arteries that are pumping blood to my hands. And in between there are capillaries. So I have a continuous system of blood flow.

We have, I have the same amount of LDL circulating in my veins. You know, you have veins on your arm too. You can see these are returning blood to my heart from my hands. Deeper in my arm, I have arteries that are pumping blood to my hands. And in between there are capillaries. So I have a continuous system of blood flow.

We have, I have the same amount of LDL circulating in my veins. You know, you have veins on your arm too. You can see these are returning blood to my heart from my hands. Deeper in my arm, I have arteries that are pumping blood to my hands. And in between there are capillaries. So I have a continuous system of blood flow.

The last time I checked my cholesterol, which was about a month ago, my LDL, I think, was 125 milligrams per deciliter. My ApoB is probably just over 100 or just below 100.

The last time I checked my cholesterol, which was about a month ago, my LDL, I think, was 125 milligrams per deciliter. My ApoB is probably just over 100 or just below 100.

The last time I checked my cholesterol, which was about a month ago, my LDL, I think, was 125 milligrams per deciliter. My ApoB is probably just over 100 or just below 100.

Yeah, somebody would consider it high. Yeah. I have the same amount of LDL circulating to my hands, moving through the capillaries, and coming back in my veins. But in me and all humans, we do not see atherosclerosis occurring in native veins. So atherosclerosis doesn't occur in a vein at all in humans. It's because it's lower pressure.