Paul Saladino

Remember, I did four years of cardiology as a PA. I gave a lot of statin prescriptions and I was proud of it at the time because I didn't know any better. So I've been in this world. We're treating the wrong thing and we're worried about the wrong things.

Remember, I did four years of cardiology as a PA. I gave a lot of statin prescriptions and I was proud of it at the time because I didn't know any better. So I've been in this world. We're treating the wrong thing and we're worried about the wrong things.

And the reason this is important to me is because people invariably get healthier when they eat more red meat, when they eat more animal fats, and when they decrease seed oils and their LDL goes up.

And the reason this is important to me is because people invariably get healthier when they eat more red meat, when they eat more animal fats, and when they decrease seed oils and their LDL goes up.

And the reason this is important to me is because people invariably get healthier when they eat more red meat, when they eat more animal fats, and when they decrease seed oils and their LDL goes up.

I think it's a centralized narrative from the 1950s, from Ancel Keys, this lipid hypothesis that is then fueled by potentially company data that are trying to muddy the waters and confuse people.

I think it's a centralized narrative from the 1950s, from Ancel Keys, this lipid hypothesis that is then fueled by potentially company data that are trying to muddy the waters and confuse people.

I think it's a centralized narrative from the 1950s, from Ancel Keys, this lipid hypothesis that is then fueled by potentially company data that are trying to muddy the waters and confuse people.

Yeah, I think so. I mean, this is very contentious in Western medicine, and there are many very intelligent, well-intentioned doctors that disagree with me and still strongly believe that LDL is causal in atherosclerosis. But when I look at the data, there are too many inconsistencies. There are studies like Dave Feldman's and Matt Budoff's.

Yeah, I think so. I mean, this is very contentious in Western medicine, and there are many very intelligent, well-intentioned doctors that disagree with me and still strongly believe that LDL is causal in atherosclerosis. But when I look at the data, there are too many inconsistencies. There are studies like Dave Feldman's and Matt Budoff's.

Yeah, I think so. I mean, this is very contentious in Western medicine, and there are many very intelligent, well-intentioned doctors that disagree with me and still strongly believe that LDL is causal in atherosclerosis. But when I look at the data, there are too many inconsistencies. There are studies like Dave Feldman's and Matt Budoff's.

There are so many examples of people with elevated LDL who do not develop atherosclerosis. And I think that it may come down to syntax and linguistics. I would agree, LDL is involved in the causal cascade. If you are insulin resistant and we lower your LDL, your risk of heart disease is unequivocally lower.

There are so many examples of people with elevated LDL who do not develop atherosclerosis. And I think that it may come down to syntax and linguistics. I would agree, LDL is involved in the causal cascade. If you are insulin resistant and we lower your LDL, your risk of heart disease is unequivocally lower.

There are so many examples of people with elevated LDL who do not develop atherosclerosis. And I think that it may come down to syntax and linguistics. I would agree, LDL is involved in the causal cascade. If you are insulin resistant and we lower your LDL, your risk of heart disease is unequivocally lower.

However, we didn't treat the real cause, which was your insulin resistance and your metabolic dysfunction. And I hope that we were honest with you about the side effects of the statins or whatever we use to lower your LDL and major questions come up, not necessarily with statins, but overall with LDL lowering about all cause mortality.

However, we didn't treat the real cause, which was your insulin resistance and your metabolic dysfunction. And I hope that we were honest with you about the side effects of the statins or whatever we use to lower your LDL and major questions come up, not necessarily with statins, but overall with LDL lowering about all cause mortality.

However, we didn't treat the real cause, which was your insulin resistance and your metabolic dysfunction. And I hope that we were honest with you about the side effects of the statins or whatever we use to lower your LDL and major questions come up, not necessarily with statins, but overall with LDL lowering about all cause mortality.

Just because you're decreasing someone's risk of cardiovascular disease death doesn't mean they're living longer or living better. Because sometimes you just shift it, right? We know that you can just shift the pathology from cardiovascular disease to dementia or to cancer or to some of these other issues. And so that's not the goal. That's not treating a human. That's not a healthy human.

Just because you're decreasing someone's risk of cardiovascular disease death doesn't mean they're living longer or living better. Because sometimes you just shift it, right? We know that you can just shift the pathology from cardiovascular disease to dementia or to cancer or to some of these other issues. And so that's not the goal. That's not treating a human. That's not a healthy human.

Just because you're decreasing someone's risk of cardiovascular disease death doesn't mean they're living longer or living better. Because sometimes you just shift it, right? We know that you can just shift the pathology from cardiovascular disease to dementia or to cancer or to some of these other issues. And so that's not the goal. That's not treating a human. That's not a healthy human.