Derek (More Plates More Dates)

you know fibrosis or undergoes fibrosis and like you're screwed in that spot essentially unless you transplant non-aga androgenic alopecia affected hair follicles into that dead zone but like you need a lot of hair to offset like a completely bald head and it's like typically not possible if you've let yourself get too far gone so

And it's really interesting too, because these hair follicles, they're not prone to the same miniaturization.

So even if you transplant it from here to here, it's not going to undergo the same effect, even though it's in that area, interestingly enough.

But these hair follicles are highly prone to miniaturization if you are susceptible to hair loss.

What makes you susceptible to hair loss?

genetics but in general are you gonna bank on you being the one guy like how many guys do you know who 50 years old plus have like no visible hair loss whatsoever and it looks like they did when they were 19 years old

Is it actually though?

Okay.

And you, you have a son you said?

Oh, he must, he's gonna be thrilled then.

Typically, it's thought to be the mom's dad.

It doesn't always play out like that.

But, like, that's a good... He might not have to take, you know, the hormone-crushing drugs.

He might be one of the fringe lucky ones.

So, proactively, as unfortunate of a reality as it is, you have to weigh the risk to reward on inhibiting DHT.

So...

How far ahead you get of this kind of impacts how intensive of a protocol you have to use as well as your susceptibility to that androgenic stimulation, which is also going to be contingent on your hormone level.

So if you're hypogonadal and then you correct that and bump yourself up to high normal, you might have just doubled your androgen load in your scalp for all you know.

And the proportional increase is like magnified multiple fold because it's

more 5-alpha reductase expression in the scalp than like anywhere else essentially.