Dr. Ben Bikman

It's ubiquitous.

You would just want to control it, I guess.

So with regards to saturated fat, my own work, when I published that paper in...

2010 maybe, I left that project with this idea that saturated fats are thus a cause of insulin resistance.

And I had to challenge my own assumptions when I saw the work of Dr. Jeff Volek, a friend and a legend in the realm of low-carbohydrate studies, because he published some incredibly compelling papers.

Over a few papers, he found that

I had to sort of challenge the model where I thought, all right, I was treating cells with saturated fat.

Is that the same as a human eating it?

And of course it's not.

And now to touch on his work, you can have humans that if the carbohydrate levels are going down, they can eat two or three or four times more saturated fat than a high carb group.

And then they're circulating levels of saturated fat.

So the saturated fat in some in the plasma is significantly lower.

That's because most of the saturated fat that's flowing through our veins is coming from the liver.

When the liver is told to make fat through de novo lipogenesis, the fat that it makes is palmitate.

So most of the saturated fat we have flowing through our blood that's going to get to a cell

is going to be coming from what the liver's making, not from what we're eating.

And he showed this very, very well.

But that's only in the background of low carbs.

Exactly.

Yeah.