Dr. Stephanie Venn-Watson

So it was just like, let's focus on these species, not to dismiss the short list, the basic science, but it's an unlock. So the seven must-haves of longevity, and this is assuming that longevity, the way we define it is as a geroprotector, so a molecule that slows our aging rate and therefore slows the onset of the chronic diseases that kill us.

healthier, and then by default, be able to extend your own longevity so that you can live longer. So of those, the seven longevity must-haves are first, this molecule needs to tap into the longevity-regulating pathway. It's this complicated keg, it's called the keg pathway, and it's based upon a hypocaloric diet.

healthier, and then by default, be able to extend your own longevity so that you can live longer. So of those, the seven longevity must-haves are first, this molecule needs to tap into the longevity-regulating pathway. It's this complicated keg, it's called the keg pathway, and it's based upon a hypocaloric diet.

healthier, and then by default, be able to extend your own longevity so that you can live longer. So of those, the seven longevity must-haves are first, this molecule needs to tap into the longevity-regulating pathway. It's this complicated keg, it's called the keg pathway, and it's based upon a hypocaloric diet.

So caloric restriction and the whole pathway that's been built out on how caloric restriction works extends longevity across multiple species. At the heart of it is activating AMPK and inhibiting mTOR, which, as you had mentioned, metformin activates AMPK and rapamycin inhibits mTOR. C15 does both. So that was really exciting to see. It's really hitting at the heart as other longevity molecules.

So caloric restriction and the whole pathway that's been built out on how caloric restriction works extends longevity across multiple species. At the heart of it is activating AMPK and inhibiting mTOR, which, as you had mentioned, metformin activates AMPK and rapamycin inhibits mTOR. C15 does both. So that was really exciting to see. It's really hitting at the heart as other longevity molecules.

So caloric restriction and the whole pathway that's been built out on how caloric restriction works extends longevity across multiple species. At the heart of it is activating AMPK and inhibiting mTOR, which, as you had mentioned, metformin activates AMPK and rapamycin inhibits mTOR. C15 does both. So that was really exciting to see. It's really hitting at the heart as other longevity molecules.

The second is that it needs to target these... And just to back up on that for a minute, because people might not understand that.

The second is that it needs to target these... And just to back up on that for a minute, because people might not understand that.

The second is that it needs to target these... And just to back up on that for a minute, because people might not understand that.

That's right.

That's right.

That's right.

That's right. That's right. Thank you for that. And your book, by the way, does a great job explaining all of those. It's hard to explain, trust me. If anyone wants to dive in more, please read your book, Young Forever. So then the second one exactly feeds into the hallmarks of aging. There's 12. The list keeps getting longer, it feels like, every couple of years.

That's right. That's right. Thank you for that. And your book, by the way, does a great job explaining all of those. It's hard to explain, trust me. If anyone wants to dive in more, please read your book, Young Forever. So then the second one exactly feeds into the hallmarks of aging. There's 12. The list keeps getting longer, it feels like, every couple of years.

That's right. That's right. Thank you for that. And your book, by the way, does a great job explaining all of those. It's hard to explain, trust me. If anyone wants to dive in more, please read your book, Young Forever. So then the second one exactly feeds into the hallmarks of aging. There's 12. The list keeps getting longer, it feels like, every couple of years.

Yeah, so there's now multiple studies showing that C15 targets six of them. And that includes helping C15 directly repairs mitochondrial function. It has a skip function where it's able to help restore broken mitochondria by enabling it to make energy. when normally it wouldn't be able to, which is exciting and important. It helps with poor cellular signaling.

Yeah, so there's now multiple studies showing that C15 targets six of them. And that includes helping C15 directly repairs mitochondrial function. It has a skip function where it's able to help restore broken mitochondria by enabling it to make energy. when normally it wouldn't be able to, which is exciting and important. It helps with poor cellular signaling.

Yeah, so there's now multiple studies showing that C15 targets six of them. And that includes helping C15 directly repairs mitochondrial function. It has a skip function where it's able to help restore broken mitochondria by enabling it to make energy. when normally it wouldn't be able to, which is exciting and important. It helps with poor cellular signaling.

We know that it not only activates AMPK and inhibits mTOR, but it also activates PPAR alpha delta and AKT, and it inhibits JAK-STAT and HDAC6. So that was a bunch of letters.