Emilia Javorsky
speaker
1,653 appearances
2 recordings
1 series
first heard Mar 2026
last heard 20 Mar
Emilia Javorsky’s voice in public audio — every appearance, attributed to the second.
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recordings per month · last 12 monthsRecordings per month over the last 12 months — 2 in all, peaking in Mar 2026 with 2.
Appearances
It's not what's the whole patient story and context around this, right?
I think the data set that is probably one of the more interesting ones that starts to solve this is the work that has been done with the UK Biobank.
So this was an effort that was started decades ago by the UK government to basically follow 500,000 people over decades and use every state-of-the-art measurement technique that came online, the imaging, the proteomics, the everything, and see what their health outcomes look like and just measure the population over time.
And so those are probably the examples, the patients within that data set that went on to develop cancer of where we have
like the best longitudinal data and pictures of patients.
So there are efforts to try and close this gap, but they are woefully sort of small and under-resourced and insufficient relative to the problem at hand.
Yes, these are all wonderful, amazing creatures.
And what they have in common is they very much have inspired scientists of, are there examples in nature of mammals that live a decently long time and don't get cancer, right?
And they all fall into this bucket.
And in fact, nature has solved this problem, right?
Right.
Nature has solved biology and saying, what are ways we can engineer biology that confer a degree of cancer resilience?
And so the idea here was like, oh, this is so great.
We have these examples of nature.
We'll go study them and they will reveal to us the way that we become cancer resistant and what that mechanism is.
And then what scientists actually found is instead of giving us this panacea or this sort of universal cancer resilience mechanism,
they kind of all developed cancer resilience through totally independent and distinct genetic pathways and had very different resilience phenotypes.
So basically, the way that they may have their cancer resilience was entirely different between each species.
And so that kind of doused our hopes that like, all right, there's not a universal mechanism here.
And the
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