Michael Levin
speaker
450 appearances
1 recordings
1 series
first heard May 2025
last heard May 2025
Michael Levin’s voice in public audio — every appearance, attributed to the second.
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Appearances
I'll just give you a couple of my my favorite examples.
Um uh this this this Fankhauser work uh was was was.
Was incredible.
So you got this newt, and uh you take a cross section through the kidney tubules, and you see there's like eight to ten cells that work together, and they give you this thing with alumen in the middle, and it's this nice, nice tubule.
So you can make these polyploid newts.
If you make polyploid newts, they have you know extra copies of of their the genetic material.
And as a result, what they do is they make their cells bigger to accommodate the bigger nucleus.
Okay, so that's cool, but the newt stays the same size.
So you say, well, how did
that happen and you take a cross-section and you see that actually, well, then there are fewer of these larger cells to build the same thing.
If you really go all the way, you know, all the way up and you make, I think there were like six N newts or something like that, you have one gigantic cell that bends around itself and leaves and leaves a hole in the middle.
Now, now that's a different mechanism of cytoskeletal bending instead of cell-to-cell communication.
And just think about what it means if you're a newt coming into the world.
What can you count on?
I mean, you know you can't really know what your environment's gonna be, but you can't even count on your own parts.
You don't know how many copies.
Copies of your genetic material you're going to have.
You don't know how big your cells are going to be, how many cells, as as Cook showed, you can, you know, take away cells, add cells.
It doesn't matter.
It'll still rescale.
Showing 201–220 of 450 · page 11 of 23
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