Athena Aktipis
speaker
1,334 appearances
1 recordings
1 series
first heard Jan 2026
last heard 9 Jan
Athena Aktipis’s voice in public audio — every appearance, attributed to the second.
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recordings per month · last 12 monthsRecordings per month over the last 12 months — 1 in all, peaking in Jan 2026 with 1.
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Yeah, exactly.
Right?
But that's the thing.
It's like we have to approach all of these terrible things with a little bit of humor.
Otherwise, how are we going to keep working on them?
So I think that one of the things that's actually missing from the way that a lot of people think about cancer, especially in sort of oncology and cancer biology, is this window of what was going on very early in multicellularity, which you made a reference to.
But
You know, if you consider, you know, like the early the early stages where you've got like some, you know, groups of cells and they're they're growing and they're, you know, producing like public goods, you know, that like are good for all the cells in there.
And if you get like a cell, you know, coming from this other group and popping in there, it can, you know, get the benefits of what, you know, that other group has created.
It's just, you know, it's a free rider problem of common pool resource problem.
It's like, you know, the sort of classic kind of.
And I think, you know, the very evolution of multicellularity is not just about regulating the cooperation within an entity that is, you know, beginning and might evolve, you know, that cheating, but the...
the early evolution of multicellularity was the evolution of prevention of contagious cancer, I think.
I think we should be thinking about that as one of the fundamental things that was going on in the early stages of multicellularity.
And so a lot of these systems that we have that are part of our immune system have many of their origins in
That original selection pressure in one way or another, or at least that's where we need to kind of start when we think about what kinds of mechanisms might have evolved that adaptive problem of, you know, cells just, you know, popping in and trying to get the benefits of that that entity.
So many of our cancer suppression systems could function.
potentially originally have been in place for prevention of contagious cancer, right?
It's an open question, like what of our cancer prevention mechanisms have to do with that versus having to do with maybe the more modern problem of preventing cancer from the inside as opposed to that hijacking from the outside.
Well, so, you know, Rob, when you like are working in like an area where you're like applying theoretical new theoretical frameworks, you're bringing things together.
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