Benjamin Thompson
speaker
746 appearances
10 recordings
1 series
first heard Jul 2026
last heard 2 Sep
Benjamin Thompson’s voice in public audio — every appearance, attributed to the second.
Trend
recordings per month · last 12 monthsRecordings per month over the last 12 months — 10 in all, peaking in Jul 2026 with 5.
Appearances
They found a strong link between the maximum lifespan and the accuracy and efficiency of double strand brake repair in skin and lung cells.
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That superior repair was explained in large part by one member of a family of enzymes called sertuins, which are known to have roles in aging, metabolism, and the stability of the genome.
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The overexpression of the sertuin cert six had already been linked to extended lifespan in mice.
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In the twenty nineteen study, the team identified five amino acids that differ between the beaver and mouse versions of cert six and seem to make the beaver version more effective.
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Beavers live for ten to twelve years in the wild, whereas mice typically live for a few years at most.
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Genetic studies suggest that some human centenarians might also carry a superior variant of the gene CERT six, says geneticist Jan Weg at the Albert Einstein College of Medicine in New York City.
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VAGE co leads a multi team effort to identify important genes and pathways in centenarians, validate them and develop drugs that target them.
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The team has identified a group of compounds called fuchoidans, which occur naturally in brown seaweed and activate the cert six protein as potential therapeutics.
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Studies by Robins, Gorbinova, and others show that supplementing mouse diets with fucoidans improves the animals' DNA repair, reduces senescence, and extends their health span and lifespan.
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Clinician geroscientist Andrea Meyer, director of the National University of Singapore's Academy for Healthy Longevity, is now leading a study that gives fucoidans to men aged fifty to eighty.
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The study is looking at cellular markers of aging and clinical outcomes, measuring as directly as possible how phicoidans affect biology.
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The ins and outs of DNA repair don't just differ from organism to organism, they also differ from cell to cell.
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Sperm and egg cells seem to accumulate much less DNA damage than other cell types do.
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DNA repair is energetically costly, so it makes sense that it would be prioritized in cells that must pass genetic information down the generations.
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Our germ cells are, in a sense, immortal, Schumacher says.
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He and his colleagues have identified a possible key to this immortality.
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In twenty twenty three, the team reported from experiments in the roundworm C elegans that a protein complex already known for its role in cell proliferation represses many DNA repair genes in non reproductive or somatic cells.
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This dream complex is found across species.
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When the team turned it off in a mouse model of a premature aging syndrome, the mice showed less DNA damage.
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Switching it off in human cells boosted the expression of DNA repair genes.
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Showing 61–80 of 746 · page 4 of 38
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