Gary Brecka

Originally used as machine lubricants, these oils were never designed for human consumption. They're a byproduct of industrial agriculture, cheap to produce, subsidized, and now they're in everything. We've gone from eating virtually zero seed oils in 1900 to consuming 30 pounds per person per year in the United States. That's not a dietary shift, it's a metabolic experiment.

And we're the test subjects. So let's address misinformation. Seed oils have long been marketed as heart healthy because they're low in saturated fat and contain omega-6 fatty acids. But what the food industry doesn't tell you is that these omega-6 polyunsaturated fatty acids, PUFAs, especially linoleic acid, are highly unstable and they're prone to oxidation.

And we're the test subjects. So let's address misinformation. Seed oils have long been marketed as heart healthy because they're low in saturated fat and contain omega-6 fatty acids. But what the food industry doesn't tell you is that these omega-6 polyunsaturated fatty acids, PUFAs, especially linoleic acid, are highly unstable and they're prone to oxidation.

And we're the test subjects. So let's address misinformation. Seed oils have long been marketed as heart healthy because they're low in saturated fat and contain omega-6 fatty acids. But what the food industry doesn't tell you is that these omega-6 polyunsaturated fatty acids, PUFAs, especially linoleic acid, are highly unstable and they're prone to oxidation.

According to the Cleveland Clinic, these oxidized fat form toxic byproducts like 4-hydroxynonanol, linked to neurodegeneration and cancer, acrolein, a pro-inflammatory compound that damages endothelial cells, the lining of your blood vessels. Advanced lipid peroxidation end products, ALEs, which impair mitochondrial function.

According to the Cleveland Clinic, these oxidized fat form toxic byproducts like 4-hydroxynonanol, linked to neurodegeneration and cancer, acrolein, a pro-inflammatory compound that damages endothelial cells, the lining of your blood vessels. Advanced lipid peroxidation end products, ALEs, which impair mitochondrial function.

According to the Cleveland Clinic, these oxidized fat form toxic byproducts like 4-hydroxynonanol, linked to neurodegeneration and cancer, acrolein, a pro-inflammatory compound that damages endothelial cells, the lining of your blood vessels. Advanced lipid peroxidation end products, ALEs, which impair mitochondrial function.

The oxidative stress from these byproducts directly correlates with insulin resistance, leaky gut, neuroinflammation, hormonal dysregulation, and accelerated aging. So what happens when you quit seed oils for 30 days? Let's break it down system by system. Number one, inflammation drops fast.

The oxidative stress from these byproducts directly correlates with insulin resistance, leaky gut, neuroinflammation, hormonal dysregulation, and accelerated aging. So what happens when you quit seed oils for 30 days? Let's break it down system by system. Number one, inflammation drops fast.

The oxidative stress from these byproducts directly correlates with insulin resistance, leaky gut, neuroinflammation, hormonal dysregulation, and accelerated aging. So what happens when you quit seed oils for 30 days? Let's break it down system by system. Number one, inflammation drops fast.

Seed oils fuel a chronic low-grade inflammatory state in the body, and linoleic acid is converted into arachidonic acid, which is a precursor to the pro-inflammatory cytokines like prostaglandins and leukotrienes. In a 2020 study published in Nutrients, found that excessive omega-6 fatty acid intake creates an omega-6 to omega-3 ratio imbalance, and this leads to systemic inflammation.

Seed oils fuel a chronic low-grade inflammatory state in the body, and linoleic acid is converted into arachidonic acid, which is a precursor to the pro-inflammatory cytokines like prostaglandins and leukotrienes. In a 2020 study published in Nutrients, found that excessive omega-6 fatty acid intake creates an omega-6 to omega-3 ratio imbalance, and this leads to systemic inflammation.

Seed oils fuel a chronic low-grade inflammatory state in the body, and linoleic acid is converted into arachidonic acid, which is a precursor to the pro-inflammatory cytokines like prostaglandins and leukotrienes. In a 2020 study published in Nutrients, found that excessive omega-6 fatty acid intake creates an omega-6 to omega-3 ratio imbalance, and this leads to systemic inflammation.

Once seed oils are removed, inflammatory markers like C-reactive protein begin to drop within weeks. Many people report improved joint pain, reduced bloating, and relief from conditions like rosacea, psoriasis, and even migraines. Your skin starts healing from the inside out. That's the second thing that you'll see.

Once seed oils are removed, inflammatory markers like C-reactive protein begin to drop within weeks. Many people report improved joint pain, reduced bloating, and relief from conditions like rosacea, psoriasis, and even migraines. Your skin starts healing from the inside out. That's the second thing that you'll see.

Once seed oils are removed, inflammatory markers like C-reactive protein begin to drop within weeks. Many people report improved joint pain, reduced bloating, and relief from conditions like rosacea, psoriasis, and even migraines. Your skin starts healing from the inside out. That's the second thing that you'll see.

Seed oils damage the phospholipid bilayer, the outer membrane of your skin cells, causing dryness, acne, and impaired barrier function. In a 2017 dermatology review, high linoleic acid intake was linked to oxidative damage in the skin, delayed wound healing, and increased sebum production.

Seed oils damage the phospholipid bilayer, the outer membrane of your skin cells, causing dryness, acne, and impaired barrier function. In a 2017 dermatology review, high linoleic acid intake was linked to oxidative damage in the skin, delayed wound healing, and increased sebum production.

Seed oils damage the phospholipid bilayer, the outer membrane of your skin cells, causing dryness, acne, and impaired barrier function. In a 2017 dermatology review, high linoleic acid intake was linked to oxidative damage in the skin, delayed wound healing, and increased sebum production.

By removing seed oils and replacing them with saturated or monounsaturated fats like tallow or olive oil, your skin begins to rehydrate, restore its elasticity, and repair itself. And number three, your mitochondria begin to breathe again. PUFAs impair mitochondrial beta-oxidation. They slow fat burning and ATP production.